Defective activation of clotting, fibrinolytic, and permeability-enhancing systems in human Fletcher trait plasma.
about
Human plasma kallikrein. A rapid purification method with high yieldInitiation of plasma prorenin activation by Hageman factor-dependent conversion of plasma prekallikrein to kallikreinThe secondary structure of human Hageman factor (factor XII) and its alteration by activating agents.Fitzgerald Trait: Deficiency of a Hitherto Unrecognized Agent, Fitzgerald Factor, Participating in Surface-Mediated Reactions of Clotting, Fibrinolysis, Generation of Kinins, and the Property of Diluted Plasma Enhancing Vascular Permeability (PF/DilPartial purification and characterization of contact activation cofactor.Characterization of a variant prekallikrein, prekallikrein Long Beach, from a family with mixed cross-reacting material-positive and cross-reacting material-negative prekallikrein deficiencyFamilial venous thrombosis.Studies on a family with combined functional deficiencies of vitamin K-dependent coagulation factorsThe activation of plasminogen by Hageman factor (Factor XII) and Hageman factor fragmentsPotentiation of the function of Hageman factor fragments by high molecular weight kininogen.Activation and function of human Hageman factor. The role of high molecular weight kininogen and prekallikrein.Prekallikrein deficiency in a kindred with kininogen deficiency and Fitzgerald trait clotting defect. Evidence that high molecular weight kininogen and prekallikrein exist as a complex in normal human plasma.Purification of high molecular weight kininogen and the role of this agent in blood coagulation.Association of factor XI and high molecular weight kininogen in human plasma.Interactions among Hageman factor, plasma prekallikrein, high molecular weight kininogen, and plasma thromboplastin antecedent.Amidolytic properties of single-chain activated Hageman factor.Role of surface in surface-dependent activation of Hageman factor (blood coagulation factor XII).FXII.Variability in purified dysfunctional C1(-)-inhibitor proteins from patients with hereditary angioneurotic edema. Functional and analytical gel studies.Granulocyte elastase cleaves human high molecular weight kininogen and destroys its clot-promoting activity.Fletcher Factor Deficiency: Report of a New Family
P2860
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P2860
Defective activation of clotting, fibrinolytic, and permeability-enhancing systems in human Fletcher trait plasma.
description
1974 nî lūn-bûn
@nan
1974年の論文
@ja
1974年学术文章
@wuu
1974年学术文章
@zh
1974年学术文章
@zh-cn
1974年学术文章
@zh-hans
1974年学术文章
@zh-my
1974年学术文章
@zh-sg
1974年學術文章
@yue
1974年學術文章
@zh-hant
name
Defective activation of clotti ...... n human Fletcher trait plasma.
@en
Defective activation of clotti ...... n human Fletcher trait plasma.
@nl
type
label
Defective activation of clotti ...... n human Fletcher trait plasma.
@en
Defective activation of clotti ...... n human Fletcher trait plasma.
@nl
prefLabel
Defective activation of clotti ...... n human Fletcher trait plasma.
@en
Defective activation of clotti ...... n human Fletcher trait plasma.
@nl
P2093
P356
P1433
P1476
Defective activation of clotti ...... n human Fletcher trait plasma.
@en
P2093
P304
P356
10.1161/01.RES.34.5.641
P577
1974-05-01T00:00:00Z