Diesel exhaust particles enhance airway responsiveness following allergen exposure in mice.
about
Diesel exhaust and asthma: hypotheses and molecular mechanisms of action.Effects of nano particles on antigen-related airway inflammation in mice.Animal models of occupational asthma: tools for understanding disease pathogenesisSuppression of the NF-κB pathway by diesel exhaust particles impairs human antimycobacterial immunityThe role of oxidative stress in ambient particulate matter-induced lung diseases and its implications in the toxicity of engineered nanoparticlesPollution and the immune response: atopic diseases--are we too dirty or too clean?Mouse models to unravel the role of inhaled pollutants on allergic sensitization and airway inflammationEffects of diesel exhaust particle exposure on a murine model of asthma due to soybeanPulmonary exposure to particles during pregnancy causes increased neonatal asthma susceptibility.Long-Term Effects of Diesel Exhaust Particles on Airway Inflammation and Remodeling in a Mouse Model.Effects of maternal exposure to di-(2-ethylhexyl) phthalate during fetal and/or neonatal periods on atopic dermatitis in male offspringImpact of diesel exhaust particles on th2 response in the lung in asthmatic mice.Immunotoxicologic analysis of maternal transmission of asthma risk.Effects of nanoparticles on lung physiology in the presence or absence of antigen.Cacao liquor proanthocyanidins inhibit lung injury induced by diesel exhaust particles.Titanium dioxide nanoparticles aggravate atopic dermatitis-like skin lesions in NC/Nga mice.Inhalation of concentrated ambient particulate matter near a heavily trafficked road stimulates antigen-induced airway responses in mice.Effects of components derived from diesel exhaust particles on lung physiology related to antigen.Unique pulmonary immunotoxicological effects of urban PM are not recapitulated solely by carbon black, diesel exhaust or coal fly ash.Inhibition of pan neurotrophin receptor p75 attenuates diesel particulate-induced enhancement of allergic airway responses in C57/B16J mice.Effect of maternal exposure to ozone on reproductive outcome and immune, inflammatory, and allergic responses in the offspring.Low-dose benzo[a]pyrene aggravates allergic airway inflammation in mice.Diesel exhaust particles enhance lung injury related to bacterial endotoxin through expression of proinflammatory cytokines, chemokines, and intercellular adhesion molecule-1.Exposure of pregnant mice to an air pollutant aerosol increases asthma susceptibility in offspring.Effects of diesel exhaust particles on antigen-presenting cells and antigen-specific Th immunity in mice.Residual oil fly ash amplifies allergic cytokines, airway responsiveness, and inflammation in mice.
P2860
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P2860
Diesel exhaust particles enhance airway responsiveness following allergen exposure in mice.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Diesel exhaust particles enhan ...... ing allergen exposure in mice.
@en
Diesel exhaust particles enhan ...... ing allergen exposure in mice.
@nl
type
label
Diesel exhaust particles enhan ...... ing allergen exposure in mice.
@en
Diesel exhaust particles enhan ...... ing allergen exposure in mice.
@nl
prefLabel
Diesel exhaust particles enhan ...... ing allergen exposure in mice.
@en
Diesel exhaust particles enhan ...... ing allergen exposure in mice.
@nl
P2093
P2860
P1476
Diesel exhaust particles enhan ...... ing allergen exposure in mice.
@en
P2093
P2860
P304
P356
10.3109/08923979809038548
P577
1998-05-01T00:00:00Z