In progressive nephropathies, overload of tubular cells with filtered proteins translates glomerular permeability dysfunction into cellular signals of interstitial inflammation.
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Angiotensin-converting enzyme inhibition prevents glomerular-tubule disconnection and atrophy in passive Heymann nephritis, an effect not observed with a calcium antagonistEffects of Reactive Oxygen Species on in vitro Filtration of Water and Albumin across Glomerular Basement MembraneLocal renal aldosterone production induces inflammation and matrix formation in kidneys of diabetic rats.Size and conformation of Ficoll as determined by size-exclusion chromatography followed by multiangle light scatteringPotential Role of Serum and Urinary Biomarkers in Diagnosis and Prognosis of Diabetic Nephropathy.The effect of progressive glomerular disease on megalin-mediated endocytosis in the kidneyUrinary protein markers predict the severity of renal histological lesions in children with mesangial proliferative glomerulonephritis.Megalin contributes to the early injury of proximal tubule cells during nonselective proteinuriaReceptor-associated protein blocks internalization and cytotoxicity of myeloma light chain in cultured human proximal tubular cells.Mitochondrial impairment in the five-sixth nephrectomy model of chronic renal failure: proteomic approach.Remission of renal disease: recounting the challenge, acquiring the goalNo evidence for statin-induced proteinuria in healthy volunteers as assessed by proteomic analysis.Complement-mediated dysfunction of glomerular filtration barrier accelerates progressive renal injury.Immune and inflammatory role in renal disease.Diabetic albuminuria is due to a small fraction of nephrons distinguished by albumin-stained tubules and glomerular adhesions.Ezetimibe and simvastatin for the prevention of cardiovascular events in predialysis chronic kidney disease patients: a review.Pathophysiology of the diabetic kidney.Tubular atrophy in the pathogenesis of chronic kidney disease progression.Just Look! Intravital Microscopy as the Best Means to Study Kidney Cell Death Dynamics.Statin treatment before percutaneous cononary intervention.Prostaglandin D2 synthase: Apoptotic factor in alzheimer plasma, inducer of reactive oxygen species, inflammatory cytokines and dialysis dementia.RAGE drives the development of glomerulosclerosis and implicates podocyte activation in the pathogenesis of diabetic nephropathy.Albuminuria--marker of progressive renal disease.
P2860
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P2860
In progressive nephropathies, overload of tubular cells with filtered proteins translates glomerular permeability dysfunction into cellular signals of interstitial inflammation.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
In progressive nephropathies, ...... of interstitial inflammation.
@en
In progressive nephropathies, ...... of interstitial inflammation.
@nl
type
label
In progressive nephropathies, ...... of interstitial inflammation.
@en
In progressive nephropathies, ...... of interstitial inflammation.
@nl
prefLabel
In progressive nephropathies, ...... of interstitial inflammation.
@en
In progressive nephropathies, ...... of interstitial inflammation.
@nl
P2093
P1476
In progressive nephropathies, ...... s of interstitial inflammation
@en
P2093
P304
P577
1998-07-01T00:00:00Z