Upregulated ank expression in osteoarthritis can promote both chondrocyte MMP-13 expression and calcification via chondrocyte extracellular PPi excess.
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Hypoxia-inducible factor regulation of ANK expression in nucleus pulposus cells: possible implications in controlling dystrophic mineralization in the intervertebral discNovel ANKH amino terminus mutation (Pro5Ser) associated with early-onset calcium pyrophosphate disease with associated phosphaturiaUnilateral anterior crossbite induces aberrant mineral deposition in degenerative temporomandibular cartilage in rats.Role of the progressive ankylosis gene (ank) in cartilage mineralization.The inorganic pyrophosphate transporter ANK preserves the differentiated phenotype of articular chondrocyte.The effects of amphiregulin induced MMP-13 production in human osteoarthritis synovial fibroblast.Physiologic and pathologic functions of the NPP nucleotide pyrophosphatase/phosphodiesterase family focusing on NPP1 in calcificationBiochemical and genetic analysis of ANK in arthritis and bone diseaseInorganic pyrophosphate generation by transforming growth factor-beta-1 is mainly dependent on ANK induction by Ras/Raf-1/extracellular signal-regulated kinase pathways in chondrocytes.Vanin-1 pantetheinase drives increased chondrogenic potential of mesenchymal precursors in ank/ank miceMatrix stiffness promotes cartilage endplate chondrocyte calcification in disc degeneration via miR-20a targeting ANKH expression.Delayed onset of changes in soma action potential genesis in nociceptive A-beta DRG neurons in vivo in a rat model of osteoarthritis.The progressive ankylosis gene product ANK regulates extracellular ATP levels in primary articular chondrocytes.FGF2 alters expression of the pyrophosphate/phosphate regulating proteins, PC-1, ANK and TNAP, in the calvarial osteoblastic cell line, MC3T3E1(C4).Developmental mechanisms in articular cartilage degradation in osteoarthritisThe Therapeutic Potential of Exogenous Adenosine Triphosphate (ATP) for Cartilage Tissue Engineering.P5L mutation in Ank results in an increase in extracellular inorganic pyrophosphate during proliferation and nonmineralizing hypertrophy in stably transduced ATDC5 cells.Oxygen tension regulates the expression of ANK (progressive ankylosis) in an HIF-1-dependent manner in growth plate chondrocytes.The role of pyrophosphate/phosphate homeostasis in terminal differentiation and apoptosis of growth plate chondrocytes.Investigating conversion of endplate chondrocytes induced by intermittent cyclic mechanical unconfined compression in three-dimensional cultures.TGF β-1 administration during ex vivo expansion of human articular chondrocytes in a serum-free medium redirects the cell phenotype toward hypertrophy.ERK-1/2 and p38 in the regulation of hypertrophic changes of normal articular cartilage chondrocytes induced by osteoarthritic subchondral osteoblasts.
P2860
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P2860
Upregulated ank expression in osteoarthritis can promote both chondrocyte MMP-13 expression and calcification via chondrocyte extracellular PPi excess.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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name
Upregulated ank expression in ...... cyte extracellular PPi excess.
@en
Upregulated ank expression in ...... cyte extracellular PPi excess.
@nl
type
label
Upregulated ank expression in ...... cyte extracellular PPi excess.
@en
Upregulated ank expression in ...... cyte extracellular PPi excess.
@nl
prefLabel
Upregulated ank expression in ...... cyte extracellular PPi excess.
@en
Upregulated ank expression in ...... cyte extracellular PPi excess.
@nl
P1476
Upregulated ank expression in ...... cyte extracellular PPi excess.
@en
P2093
R Terkeltaub
P304
P356
10.1016/J.JOCA.2003.12.004
P577
2004-04-01T00:00:00Z