Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent.
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Control of autoimmune CNS inflammation by astrocytesThe role of CXCR3 in DSS-induced colitis.Deletion of astroglial CXCL10 delays clinical onset but does not affect progressive axon loss in a murine autoimmune multiple sclerosis modelChemokine receptor expression by inflammatory T cells in EAE.Anti-inflammatory mechanisms of IFN-γ studied in experimental autoimmune encephalomyelitis reveal neutrophils as a potential target in multiple sclerosis.Multiple sclerosis-induced neuropathic pain: pharmacological management and pathophysiological insights from rodent EAE modelsMurine Cytomegalovirus Infection Induces Susceptibility to EAE in Resistant BALB/c Mice.CXCR3 signaling in glial cells ameliorates experimental autoimmune encephalomyelitis by restraining the generation of a pro-Th17 cytokine milieu and reducing CNS-infiltrating Th17 cells.Neuropilin-1 modulates interferon-γ-stimulated signaling in brain microvascular endothelial cells.T-bet promotes the accumulation of encephalitogenic Th17 cells in the CNS.
P2860
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P2860
Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年學術文章
@zh-hant
name
Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent.
@en
Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent.
@nl
type
label
Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent.
@en
Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent.
@nl
prefLabel
Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent.
@en
Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent.
@nl
P2860
P356
P1476
Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent.
@en
P2860
P304
P356
10.1002/EJI.201343499
P407
P577
2013-08-21T00:00:00Z