Tunicamycin potentiates paclitaxel-induced apoptosis through inhibition of PI3K/AKT and MAPK pathways in breast cancer.

about

Protein Kinase Targets in Breast Cancer.

P2860

Q47351355-8D3003A8-3752-4EBD-9EF9-6EBD1C0A9174

P2860

Tunicamycin potentiates paclitaxel-induced apoptosis through inhibition of PI3K/AKT and MAPK pathways in breast cancer.

Item

http://www.wikidata.org/entity/Q47988571

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Tunicamycin potentiates paclit ...... APK pathways in breast cancer.

@en

Tunicamycin potentiates paclit ...... APK pathways in breast cancer.

@nl

type

Item

label

Tunicamycin potentiates paclit ...... APK pathways in breast cancer.

@en

Tunicamycin potentiates paclit ...... APK pathways in breast cancer.

@nl

prefLabel

Tunicamycin potentiates paclit ...... APK pathways in breast cancer.

@en

Tunicamycin potentiates paclit ...... APK pathways in breast cancer.

@nl

P1476

Tunicamycin potentiates paclit ...... MAPK pathways in breast cancer

@en

P2093

Dandan Wang

http://www.w3.org/2001/XMLSchema#string

Di Wang

http://www.w3.org/2001/XMLSchema#string

Muhammad Ijaz

http://www.w3.org/2001/XMLSchema#string

Shengshi Huang

http://www.w3.org/2001/XMLSchema#string

Shu Zhang

http://www.w3.org/2001/XMLSchema#string

Xiaoyan Huang

http://www.w3.org/2001/XMLSchema#string

P2860

Conserved oncogenic behavior of the FAM83 family regulates MAPK signaling in human cancer

Regulatory Roles of MAPK Phosphatases in Cancer

PI3K inhibitors as new cancer therapeutics: implications for clinical trial design

Paclitaxel Through the Ages of Anticancer Therapy: Exploring Its Role in Chemoresistance and Radiation Therapy

Hyperactive Ras in developmental disorders and cancer

Implication of the Akt2/survivin pathway as a critical target in paclitaxel treatment in human ovarian cancer cells.

Inhibition of N-linked glycosylation impairs ALK phosphorylation and disrupts pro-survival signaling in neuroblastoma cell lines.

Time series proteome profiling to study endoplasmic reticulum stress response.

Antitumor activity of Triolimus: a novel multidrug-loaded micelle containing Paclitaxel, Rapamycin, and 17-AAG.

p53-independent apoptosis induced by paclitaxel through an indirect mechanism.

P2888

s00280-017-3393-7

P304

685-696

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1007/S00280-017-3393-7

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Yikang Shi

P577

2017-08-04T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28779263

http://www.w3.org/2001/XMLSchema#string

Tunicamycin potentiates paclitaxel-induced apoptosis through inhibition of PI3K/AKT and MAPK pathways in breast cancer.

about

P2860

P2860

Tunicamycin potentiates paclitaxel-induced apoptosis through inhibition of PI3K/AKT and MAPK pathways in breast cancer.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P50

P577

P698

P356

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P50

P577

P698