Decay-accelerating factor deficiency increases susceptibility to dextran sulfate sodium-induced colitis: role for complement in inflammatory bowel disease.
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CD97 neutralisation increases resistance to collagen-induced arthritis in miceThe C5a receptor antagonist PMX205 ameliorates experimentally induced colitis associated with increased IL-4 and IL-10A novel role for CD55 in granulocyte homeostasis and anti-bacterial host defenseDeletion of either CD55 or CD97 ameliorates arthritis in mouse modelsA demonstration and findings of a statistical approach through reanalysis of inflammatory bowel disease data.Discovery, linkage disequilibrium and association analyses of polymorphisms of the immune complement inhibitor, decay-accelerating factor gene (DAF/CD55) in type 1 diabetesEffects of huoxiangzhengqi liquid on enteric mucosal immune responses in mice with Bacillus dysenteriae and Salmonella typhimurium induced diarrheaImmune cell-derived c3 is required for autoimmune diabetes induced by multiple low doses of streptozotocin.The complement anaphylatoxin C3a receptor (C3aR) contributes to the inflammatory response in dextran sulfate sodium (DSS)-induced colitis in mice.Decay-accelerating factor ameliorates systemic autoimmune disease in MRL/lpr mice via both complement-dependent and -independent mechanismsComplement-dependent injury and protection in a murine model of acute dextran sulfate sodium-induced colitisCase report and literature review: transient Inab phenotype and an agglutinating anti-IFC in a patient with a gastrointestinal problem.Complement: coming full circle.The complement system in inflammatory bowel disease.Systematic analysis of chromatin interactions at disease associated loci links novel candidate genes to inflammatory bowel disease.Regulation of the alternative pathway of complement modulates injury and immunity in a chronic model of dextran sulphate sodium-induced colitis.Neutralization of complement component C5 ameliorates the development of dextran sulfate sodium (DSS)-colitis in mice.Depletion of complement does not impact initiation of xenobiotic-induced autoimmune disease.Suppression of inflammatory cytokine production and oxidative stress by CO-releasing molecules-liberated CO in the small intestine of thermally-injured mice.A novel protocol allowing oral delivery of a protein complement inhibitor that subsequently targets to inflamed colon mucosa and ameliorates murine colitis.
P2860
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P2860
Decay-accelerating factor deficiency increases susceptibility to dextran sulfate sodium-induced colitis: role for complement in inflammatory bowel disease.
description
2004 nî lūn-bûn
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2004年の論文
@ja
2004年学术文章
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2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Decay-accelerating factor defi ...... in inflammatory bowel disease.
@en
Decay-accelerating factor defi ...... in inflammatory bowel disease.
@nl
type
label
Decay-accelerating factor defi ...... in inflammatory bowel disease.
@en
Decay-accelerating factor defi ...... in inflammatory bowel disease.
@nl
prefLabel
Decay-accelerating factor defi ...... in inflammatory bowel disease.
@en
Decay-accelerating factor defi ...... in inflammatory bowel disease.
@nl
P2093
P1476
Decay-accelerating factor defi ...... in inflammatory bowel disease.
@en
P2093
Alan D Levine
David Spencer
Denise A Hatala
M Edward Medof
P304
P356
10.4049/JIMMUNOL.172.6.3836
P407
P577
2004-03-01T00:00:00Z