Murine CD93 (C1qRp) contributes to the removal of apoptotic cells in vivo but is not required for C1q-mediated enhancement of phagocytosis.
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Modulated interaction of the ERM protein, moesin, with CD93CD93 interacts with the PDZ domain-containing adaptor protein GIPC: implications in the modulation of phagocytosisCD93 is required for maintenance of antibody secretion and persistence of plasma cells in the bone marrow nicheA novel CD93 polymorphism in non-obese diabetic (NOD) and NZB/W F1 mice is linked to a CD4+ iNKT cell deficient stateThe pericyte and stromal cell marker CD248 (endosialin) is required for efficient lymph node expansionComplement: a key system for immune surveillance and homeostasisDBA/2J Haplotype on Distal Chromosome 2 Reduces Mertk Expression, Restricts Efferocytosis, and Increases Susceptibility to Atherosclerosis.Functional investigations on human mesenchymal stem cells exposed to magnetic fields and labeled with clinically approved iron nanoparticlesThe characterization of a novel monoclonal antibody against CD93 unveils a new antiangiogenic target.IFN priming is necessary but not sufficient to turn on a migratory dendritic cell program in lupus monocytes.Anti-nucleocapsid protein immune responses counteract pathogenic effects of Rift Valley fever virus infection in mice.Tyro3 receptor tyrosine kinases in the heterogeneity of apoptotic cell uptake.The macrophage and the apoptotic cell: an innate immune interaction viewed simplistically?The epidermal growth factor-like domain of CD93 is a potent angiogenic factor.Phagocytosis is the main CR3-mediated function affected by the lupus-associated variant of CD11b in human myeloid cellsC1q deficiency leads to the defective suppression of IFN-alpha in response to nucleoprotein containing immune complexes.CD93 and GIPC expression and localization during central nervous system inflammation.Mannose-binding lectin and innate immunity.Elevated alpha-synuclein impairs innate immune cell function and provides a potential peripheral biomarker for Parkinson's disease.Identification of aortic arch-specific quantitative trait loci for atherosclerosis by an intercross of DBA/2J and 129S6 apolipoprotein E-deficient miceAntibody-enhanced cross-presentation of self antigen breaks T cell tolerance.CD93 gene polymorphism is associated with disseminated colorectal cancer.Complement protein C1q directs macrophage polarization and limits inflammasome activity during the uptake of apoptotic cells.C1q governs deposition of circulating immune complexes and leukocyte Fcgamma receptors mediate subsequent neutrophil recruitment.Crosstalk between the alpha2beta1 integrin and c-met/HGF-R regulates innate immunityPhagocytosis of apoptotic cells and immune regulation.Hypercholesterolemia potentiates aortic endothelial response to inhaled diesel exhaustSoluble CD93 as a Novel Biomarker in Asthma ExacerbationAutophagy at the gut interface: mucosal responses to stress and the consequences for inflammatory bowel diseasesSoluble CD93 in Serum as a Marker of Allergic Inflammation.Membrane-associated CD93 regulates leukocyte migration and C1q-hemolytic activity during murine peritonitis.Complement in removal of the dead - balancing inflammation.Structure, genetics and function of the pulmonary associated surfactant proteins A and D: The extra-pulmonary role of these C type lectins.C1q: A fresh look upon an old molecule.Proteomics of Human Dendritic Cell Subsets Reveals Subset-Specific Surface Markers and Differential Inflammasome Function.Flow cytometric identification of CD93 expression on naive T lymphocytes (CD4(+)CD45RA (+) cells) in human neonatal umbilical cord blood.Detection and characterization of soluble CD93 released during inflammation.Murine low-density lipoprotein receptor-related protein 1 (LRP) is required for phagocytosis of targets bearing LRP ligands but is not required for C1q-triggered enhancement of phagocytosis.Decrease in CD93 (C1qRp) expression in a human monocyte-like cell line (U937) treated with various apoptosis-inducing chemical substances.Regulation of CD93 cell surface expression by protein kinase C isoenzymes.
P2860
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P2860
Murine CD93 (C1qRp) contributes to the removal of apoptotic cells in vivo but is not required for C1q-mediated enhancement of phagocytosis.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
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2004年学术文章
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2004年學術文章
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name
Murine CD93 (C1qRp) contribute ...... d enhancement of phagocytosis.
@en
Murine CD93
@nl
type
label
Murine CD93 (C1qRp) contribute ...... d enhancement of phagocytosis.
@en
Murine CD93
@nl
prefLabel
Murine CD93 (C1qRp) contribute ...... d enhancement of phagocytosis.
@en
Murine CD93
@nl
P2093
P50
P1476
Murine CD93 (C1qRp) contribute ...... d enhancement of phagocytosis.
@en
P2093
Anne E Bygrave
Josefina Cortes-Hernandez
Liliane Fossati-Jimack
Marina Botto
Peter J Norsworthy
Philip R Taylor
Sussan Nourshargh
P304
P356
10.4049/JIMMUNOL.172.6.3406
P407
P577
2004-03-01T00:00:00Z