The paradox of viable sup45 STOP mutations: a necessary equilibrium between translational readthrough, activity and stability of the protein.
about
Autoregulatory systems controlling translation factor expression: thermostat-like control of translational accuracy.Distinct mechanisms of mutant huntingtin toxicity in different yeast strains.Regulation of release factor expression using a translational negative feedback loop: a systems analysis.SFP1-mediated prion-dependent lethality is caused by increased Sup35 aggregation and alleviated by Sis1.
P2860
The paradox of viable sup45 STOP mutations: a necessary equilibrium between translational readthrough, activity and stability of the protein.
description
2009 nî lūn-bûn
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2009年の論文
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2009年学术文章
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2009年学术文章
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2009年学术文章
@zh-cn
2009年学术文章
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2009年学术文章
@zh-my
2009年学术文章
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2009年學術文章
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2009年學術文章
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name
The paradox of viable sup45 ST ...... and stability of the protein.
@en
The paradox of viable sup45 ST ...... and stability of the protein.
@nl
type
label
The paradox of viable sup45 ST ...... and stability of the protein.
@en
The paradox of viable sup45 ST ...... and stability of the protein.
@nl
prefLabel
The paradox of viable sup45 ST ...... and stability of the protein.
@en
The paradox of viable sup45 ST ...... and stability of the protein.
@nl
P2093
P2860
P50
P1476
The paradox of viable sup45 ST ...... and stability of the protein.
@en
P2093
Agnès Baudin-Baillieu
Denis Kiktev
Jean-Pierre Rousset
P2860
P2888
P356
10.1007/S00438-009-0447-5
P577
2009-04-16T00:00:00Z