Splenectomy protects aged mice from injury after experimental stroke.

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Myeloid-specific TAK1 deletion results in reduced brain monocyte infiltration and improved outcomes after stroke.The splenic response to stroke: from rodents to stroke subjects.

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Splenectomy protects aged mice from injury after experimental stroke.

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2017 nî lūn-bûn

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2017年学术文章

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Splenectomy protects aged mice from injury after experimental stroke.

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Splenectomy protects aged mice from injury after experimental stroke.

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Splenectomy protects aged mice from injury after experimental stroke.

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Splenectomy protects aged mice from injury after experimental stroke.

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Splenectomy protects aged mice from injury after experimental stroke.

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Splenectomy protects aged mice from injury after experimental stroke.

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Splenectomy protects aged mice from injury after experimental stroke.

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Abdullah Al Mamun

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Anjali Chauhan

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Gabriel Spiegel

http://www.w3.org/2001/XMLSchema#string

Liang Zhu

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Louise D McCullough

http://www.w3.org/2001/XMLSchema#string

Nia Harris

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P2860

The ageing systemic milieu negatively regulates neurogenesis and cognitive function.

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Vascular and neurogenic rejuvenation of the aging mouse brain by young systemic factors

Differential effects of aging and sex on stroke induced inflammation across the lifespan

Macrophage plasticity and polarization: in vivo veritas

The immunopathogenesis of sepsis

Monocyte Subsets Coregulate Inflammatory Responses by Integrated Signaling through TNF and IL-6 at the Endothelial Cell Interface.

The spleen contributes to stroke-induced neurodegeneration.

Neutrophil-mediated IL-6 receptor trans-signaling and the risk of chronic obstructive pulmonary disease and asthma.

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102-111

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scholarly article

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10.1016/J.NEUROBIOLAGING.2017.09.022

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2017-09-28T00:00:00Z

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29059593

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Splenectomy protects aged mice from injury after experimental stroke.

about

P2860

P2860

Splenectomy protects aged mice from injury after experimental stroke.

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