Parasite-specific IgM plays a significant role in the protective immune response to asexual erythrocytic stage Plasmodium chabaudi AS infection.

Parasite-specific IgM plays a significant role in the protective immune response to asexual erythrocytic stage Plasmodium chabaudi AS infection.

Item
http://www.wikidata.org/entity/Q48011236
IgM production by bone marrow plasmablasts contributes to long-term protection against intracellular bacterial infectionHost control of malaria infections: constraints on immune and erythropoeitic response kineticsIdentification of circulating biomarkers in sera of Plasmodium knowlesi-infected malaria patients--comparison against Plasmodium vivax infection.Genetic Resistance to Malaria Is Associated With Greater Enhancement of Immunoglobulin (Ig)M Than IgG Responses to a Broad Array of Plasmodium falciparum Antigens.Low antibodies against Plasmodium falciparum and imbalanced pro-inflammatory cytokines are associated with severe malaria in Mozambican children: a case-control studySuppression of Plasmodium falciparum by serum collected from a case of Plasmodium vivax infection.Identification of residues in the Cmu4 domain of polymeric IgM essential for interaction with Plasmodium falciparum erythrocyte membrane protein 1 (PfEMP1).Somatically Hypermutated Plasmodium-Specific IgM(+) Memory B Cells Are Rapid, Plastic, Early Responders upon Malaria RechallengeQuantifying variation in the potential for antibody-mediated apparent competition among nine genotypes of the rodent malaria parasite Plasmodium chabaudi.IgM in microbial infections: taken for granted?Fc-receptors and immunity to malaria: from models to vaccines.sIgM-FcμR Interactions Regulate Early B Cell Activation and Plasma Cell Development after Influenza Virus Infection.IL-27 receptor signaling regulates memory CD4+ T cell populations and suppresses rapid inflammatory responses during secondary malaria infection.Small molecule-based inhibition of MEK1/2 proteins dampens inflammatory responses to malaria, reduces parasite load, and mitigates pathogenic outcomes.Acute Disruption of Bone Marrow B Lymphopoiesis and Apoptosis of Transitional and Marginal Zone B Cells in the Spleen following a Blood-Stage Plasmodium chabaudi Infection in Mice.Effects of CD4(+)CD25(+)Foxp3(+)regulatory T cells on early Plasmodium yoelii 17XL infection in BALB/c mice.CD28 deficiency leads to accumulation of germinal-center independent IgM+ experienced B cells and to production of protective IgM during experimental malaria
P2860

Parasite-specific IgM plays a significant role in the protective immune response to asexual erythrocytic stage Plasmodium chabaudi AS infection.

Item
http://www.wikidata.org/entity/Q48011236
description
2005 nî lūn-bûn
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2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Parasite-specific IgM plays a ...... smodium chabaudi AS infection.
@en
Parasite-specific IgM plays a ...... smodium chabaudi AS infection.
@nl
type
label
Parasite-specific IgM plays a ...... smodium chabaudi AS infection.
@en
Parasite-specific IgM plays a ...... smodium chabaudi AS infection.
@nl
prefLabel
Parasite-specific IgM plays a ...... smodium chabaudi AS infection.
@en
Parasite-specific IgM plays a ...... smodium chabaudi AS infection.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P50
P577
P5875
P698
P921
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P1433
P1476
Parasite-specific IgM plays a ...... smodium chabaudi AS infection.
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P2093
P2860
P304
P31
P356
10.1111/J.1365-3024.2005.00760.X
P433
P478
P50
P577
P5875
P698
P921