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Th17 cells promote autoimmune anti-myeloperoxidase glomerulonephritis.Toll-like receptor 9 enhances nephritogenic immunity and glomerular leukocyte recruitment, exacerbating experimental crescentic glomerulonephritis.The IL-27 receptor has biphasic effects in crescentic glomerulonephritis mediated through Th1 responses.The immunodominant myeloperoxidase T-cell epitope induces local cell-mediated injury in antimyeloperoxidase glomerulonephritis.Mast cells contribute to peripheral tolerance and attenuate autoimmune vasculitis.Myeloperoxidase Peptide-Based Nasal Tolerance in Experimental ANCA-Associated GN.Endogenous Toll-Like Receptor 9 Regulates AKI by Promoting Regulatory T Cell RecruitmentThe HLA-DRB1*15:01-restricted Goodpasture's T cell epitope induces GN.Thymic deletion and regulatory T cells prevent antimyeloperoxidase GN.Mast Cell Stabilization Ameliorates Autoimmune Anti-Myeloperoxidase GlomerulonephritisAdvances in the pathogenesis of Goodpasture's disease: from epitopes to autoantibodies to effector T cells.T cell mediated autoimmune glomerular disease in mice.Mouse models of anti-neutrophil cytoplasmic antibody-associated vasculitis.The NLRP3 inflammasome in kidney disease and autoimmunity.Intrinsic renal cell and leukocyte-derived TLR4 aggravate experimental anti-MPO glomerulonephritis.Deficiency of annexin A1 in CD4+ T cells exacerbates T cell-dependent inflammation.FcγRIIB regulates T-cell autoreactivity, ANCA production, and neutrophil activation to suppress anti-myeloperoxidase glomerulonephritis.Antimyeloperoxidase antibodies rapidly induce alpha-4-integrin-dependent glomerular neutrophil adhesion.Programmed death 1 and its ligands do not limit experimental foreign antigen-induced immune complex glomerulonephritis.Myeloperoxidase (MPO)-specific CD4+ T cells contribute to MPO-anti-neutrophil cytoplasmic antibody (ANCA) associated glomerulonephritis.CD8+ T Cells Effect Glomerular Injury in Experimental Anti-Myeloperoxidase GN.Mast cell activation and degranulation promotes renal fibrosis in experimental unilateral ureteric obstruction.Renal dendritic cells adopt a pro-inflammatory phenotype in obstructive uropathy to activate T cells but do not directly contribute to fibrosisToll-like receptor 2 induces Th17 myeloperoxidase autoimmunity while Toll-like receptor 9 drives Th1 autoimmunity in murine vasculitisEndogenous foxp3(+) T-regulatory cells suppress anti-glomerular basement membrane nephritisAnti-neutrophil cytoplasmic antibodies and effector CD4+ cells play nonredundant roles in anti-myeloperoxidase crescentic glomerulonephritisTLR9 and TLR4 are required for the development of autoimmunity and lupus nephritis in pristane nephropathyMultiphoton imaging reveals a new leukocyte recruitment paradigm in the glomerulusCD4+ Th1 cells are effectors in lupus nephritis--but what are their targets?Corrigendum: Multiphoton imaging reveals a new leukocyte recruitment paradigm in the glomerulus
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description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Joshua D Ooi
@nl
Joshua D Ooi
@sl
Joshua D. Ooi
@en
Joshua D. Ooi
@es
type
label
Joshua D Ooi
@nl
Joshua D Ooi
@sl
Joshua D. Ooi
@en
Joshua D. Ooi
@es
prefLabel
Joshua D Ooi
@nl
Joshua D Ooi
@sl
Joshua D. Ooi
@en
Joshua D. Ooi
@es
P106
P1153
11540952300
P21
P31
P496
0000-0003-3328-2561