An Essential Postdevelopmental Role for Lis1 in Mice.

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An Essential Postdevelopmental Role for Lis1 in Mice.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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2018年學術文章

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An Essential Postdevelopmental Role for Lis1 in Mice.

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An Essential Postdevelopmental Role for Lis1 in Mice.

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An Essential Postdevelopmental Role for Lis1 in Mice.

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An Essential Postdevelopmental Role for Lis1 in Mice.

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An Essential Postdevelopmental Role for Lis1 in Mice.

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An Essential Postdevelopmental Role for Lis1 in Mice.

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P1476

An Essential Postdevelopmental Role for Lis1 in Mice.

@en

P2093

Jill R Turner

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Meghann M Lange

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Subhshri Sahu

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Xu Gao

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P2860

Exome sequencing identifies a DYNC1H1 mutation in a large pedigree with dominant axonal Charcot-Marie-Tooth disease

Molecular defects in the motor adaptor BICD2 cause proximal spinal muscular atrophy with autosomal-dominant inheritance

A new look at an old drug: neuroprotective effects and therapeutic potentials of lithium salts

Sudden death following selective neuronal lesions in the rat nucleus tractus solitarii

Lis1 acts as a "clutch" between the ATPase and microtubule-binding domains of the dynein motor.

Genetic Basis of Brain Malformations

Mutations in the tail domain of DYNC1H1 cause dominant spinal muscular atrophy.

Lis1 and Ndel1 influence the timing of nuclear envelope breakdown in neural stem cells.

Lis1 controls dynamics of neuronal filopodia and spines to impact synaptogenesis and social behaviour

A novel neurological phenotype in mice lacking mitochondrial manganese superoxide dismutase

P31

scholarly article

P356

10.1523/ENEURO.0350-17.2018

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P433

http://www.w3.org/2001/XMLSchema#string

P478

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P50

Deanna S Smith

Timothy Hines

Jeffery L Twiss

P577

2018-01-01T00:00:00Z

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P698

29404402

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P932

5797476

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