Stathmin/Op18 depletion induces genomic instability and leads to premature senescence in human normal fibroblasts.

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Stathmin/Op18 depletion induces genomic instability and leads to premature senescence in human normal fibroblasts.

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2017 nî lūn-bûn

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Stathmin/Op18 depletion induce ...... e in human normal fibroblasts.

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Stathmin/Op18 depletion induce ...... e in human normal fibroblasts.

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Stathmin/Op18 depletion induce ...... e in human normal fibroblasts.

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Stathmin/Op18 depletion induce ...... e in human normal fibroblasts.

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Stathmin/Op18 depletion induce ...... e in human normal fibroblasts.

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Stathmin/Op18 depletion induce ...... e in human normal fibroblasts.

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P1476

Stathmin/Op18 depletion induce ...... ce in human normal fibroblasts

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P2093

Deepmala Shrestha

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Namil Kim

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P2860

Control of microtubule dynamics by oncoprotein 18: dissection of the regulatory role of multisite phosphorylation during mitosis

ING2 regulates the onset of replicative senescence by induction of p300-dependent p53 acetylation

Recent findings and future directions for interpolar mitotic kinesin inhibitors in cancer therapy

Stathmin-dependent molecular targeting therapy for malignant tumor: the latest 5 years' discoveries and developments

A novel assay for apoptosis. Flow cytometric detection of phosphatidylserine expression on early apoptotic cells using fluorescein labelled Annexin V

A versatile viral system for expression and depletion of proteins in mammalian cells

Foxp3 expression in p53-dependent DNA damage responses

CENP-A reduction induces a p53-dependent cellular senescence response to protect cells from executing defective mitoses.

Spindle assembly and the art of regulating microtubule dynamics by MAPs and Stathmin/Op18.

The essence of senescence.

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2381-2395

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scholarly article

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10.1002/JCB.26401

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119

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Kiwon Song

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2017-10-18T00:00:00Z

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28885720

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