High cyclophilin D content of synaptic mitochondria results in increased vulnerability to permeability transition.
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Mitochondria in motor nerve terminals: function in health and in mutant superoxide dismutase 1 mouse models of familial ALSThe cellular and compartmental profile of mouse retinal glycolysis, tricarboxylic acid cycle, oxidative phosphorylation, and ~P transferring kinasesBcl-xL-mediated remodeling of rod and cone synaptic mitochondria after postnatal lead exposure: electron microscopy, tomography and oxygen consumptionMorphological and bioenergetic demands underlying the mitophagy in post-mitotic neurons: the pink-parkin pathwayCalpain 1 and Calpastatin expression is developmentally regulated in rat brainMitochondrial viability in mouse and human postmortem brainQuantitative proteomics of synaptic and nonsynaptic mitochondria: insights for synaptic mitochondrial vulnerability.Oxidative stress in the progression of Alzheimer disease in the frontal cortex.Age-related changes in mitochondrial respiration and oxidative damage in the cerebral cortex of the Fischer 344 rat.Different redox states in malignant and nonmalignant esophageal epithelial cells and differential cytotoxic responses to bile acid and honokiol.Potential therapeutic benefits of strategies directed to mitochondria.Age-related mitochondrial changes after traumatic brain injuryChronic ethanol consumption enhances sensitivity to Ca(2+)-mediated opening of the mitochondrial permeability transition pore and increases cyclophilin D in liver.Early deficits in synaptic mitochondria in an Alzheimer's disease mouse modelReperfusion promotes mitochondrial dysfunction following focal cerebral ischemia in rats.Mitochondrial and Cell Death Mechanisms in Neurodegenerative Diseases.Enhancement of anxiety, facilitation of avoidance behavior, and occurrence of adult-onset obesity in mice lacking mitochondrial cyclophilin DImmunophilin deficiency augments Ca2+-dependent glutamate release from mouse cortical astrocytes.Repetitive nerve stimulation transiently opens the mitochondrial permeability transition pore in motor nerve terminals of symptomatic mutant SOD1 miceMitochondria, Amyloid β, and Alzheimer's DiseaseLipidomic analysis and electron transport chain activities in C57BL/6J mouse brain mitochondria.Activation of the mitochondrial permeability transition pore modulates Ca2+ responses to physiological stimuli in adult neurons.Cyclophilin D deficiency improves mitochondrial function and learning/memory in aging Alzheimer disease mouse modelSynaptic mitochondrial pathology in Alzheimer's diseaseWnt Signaling Prevents the Aβ Oligomer-Induced Mitochondrial Permeability Transition Pore Opening Preserving Mitochondrial Structure in Hippocampal Neurons.Biology of mitochondria in neurodegenerative diseases.Analysis of regional brain mitochondrial bioenergetics and susceptibility to mitochondrial inhibition utilizing a microplate based systemP2Y1R-initiated, IP3R-dependent stimulation of astrocyte mitochondrial metabolism reduces and partially reverses ischemic neuronal damage in mouse.Mitochondrial DNA oxidative damage and repair in aging and Alzheimer's disease.Preparation of purified perikaryal and synaptosomal mitochondrial fractions from relatively small hypothalamic brain samplesMitochondrial membrane potential in axons increases with local nerve growth factor or semaphorin signaling.NR2A and NR2B subunits differentially mediate MAP kinase signaling and mitochondrial morphology following excitotoxic insult.Knockout of Cyclophilin-D Provides Partial Amelioration of Intrinsic and Synaptic Properties Altered by Mild Traumatic Brain Injury.Cyclophilin D is expressed predominantly in mitochondria of gamma-aminobutyric acidergic interneurons.Impaired regulation of brain mitochondria by extramitochondrial Ca2+ in transgenic Huntington disease ratsThe mitochondrial permeability transition pore in motor neurons: involvement in the pathobiology of ALS mice.Amyloid beta, mitochondrial structural and functional dynamics in Alzheimer's disease.Mitochondrial damage: a target for new therapeutic horizons.Mitochondrial calcium function and dysfunction in the central nervous system.Cyclophilin D deficiency attenuates mitochondrial and neuronal perturbation and ameliorates learning and memory in Alzheimer's disease.
P2860
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P2860
High cyclophilin D content of synaptic mitochondria results in increased vulnerability to permeability transition.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
High cyclophilin D content of ...... ty to permeability transition.
@en
High cyclophilin D content of ...... ty to permeability transition.
@nl
type
label
High cyclophilin D content of ...... ty to permeability transition.
@en
High cyclophilin D content of ...... ty to permeability transition.
@nl
prefLabel
High cyclophilin D content of ...... ty to permeability transition.
@en
High cyclophilin D content of ...... ty to permeability transition.
@nl
P2093
P1476
High cyclophilin D content of ...... ity to permeability transition
@en
P2093
James W Geddes
Kranthi Kumari Naga
Patrick G Sullivan
P304
P356
10.1523/JNEUROSCI.0646-07.2007
P407
P577
2007-07-01T00:00:00Z