Cellular and molecular mechanisms of glial scarring and progressive cavitation: in vivo and in vitro analysis of inflammation-induced secondary injury after CNS trauma.
about
Human neural stem cells differentiate and promote locomotor recovery in an early chronic spinal cord injury NOD-scid mouse modelMatrices with compliance comparable to that of brain tissue select neuronal over glial growth in mixed cortical cultures.Macrophages promote axon regeneration with concurrent neurotoxicityDisruption of spinal cord white matter and sciatic nerve geometry inhibits axonal growth in vitro in the absence of glial scarringEffects of lipopolysaccharide-induced inflammation on expression of growth-associated genes by corticospinal neurons.Cell therapy in spinal cord injury: a mini- reivewNeed for a paradigm shift in therapeutic approaches to CNS injuryNon-mammalian model systems for studying neuro-immune interactions after spinal cord injuryIntravenous multipotent adult progenitor cell treatment decreases inflammation leading to functional recovery following spinal cord injury.Arylsulfatase B improves locomotor function after mouse spinal cord injuryCharacterization of inflammatory gene expression and galectin-3 function after spinal cord injury in mice.In-vitro activation of complement system by lactic acidosis in newborn and adultsBlockade of P2 nucleotide receptors after spinal cord injury reduced the gliotic response and spared tissueOlprinone attenuates the acute inflammatory response and apoptosis after spinal cord trauma in miceInhibition of EGFR/MAPK signaling reduces microglial inflammatory response and the associated secondary damage in rats after spinal cord injuryOlfactory Ensheathing Cell Transplantation after a Complete Spinal Cord Transection Mediates Neuroprotective and Immunomodulatory Mechanisms to Facilitate RegenerationFunctional recovery following traumatic spinal cord injury mediated by a unique polymer scaffold seeded with neural stem cells.Another barrier to regeneration in the CNS: activated macrophages induce extensive retraction of dystrophic axons through direct physical interactions.Quantitative analysis by in vivo imaging of the dynamics of vascular and axonal networks in injured mouse spinal cord.Adult NG2+ cells are permissive to neurite outgrowth and stabilize sensory axons during macrophage-induced axonal dieback after spinal cord injury.Meningeal cells and glia establish a permissive environment for axon regeneration after spinal cord injury in newts.Beneficial compaction of spinal cord lesion by migrating astrocytes through glycogen synthase kinase-3 inhibition.Multipotent adult progenitor cells prevent macrophage-mediated axonal dieback and promote regrowth after spinal cord injury.Tissue sparing, behavioral recovery, supraspinal axonal sparing/regeneration following sub-acute glial transplantation in a model of spinal cord contusion.Crmp4 deletion promotes recovery from spinal cord injury by neuroprotection and limited scar formation.Assessment of gliosis around moveable implants in the brainThe complement cascade: Yin-Yang in neuroinflammation--neuro-protection and -degeneration.Functional axonal regeneration through astrocytic scar genetically modified to digest chondroitin sulfate proteoglycans.Fibrinogen triggers astrocyte scar formation by promoting the availability of active TGF-beta after vascular damageSoluble factor effects on glial cell reactivity at the surface of gel-coated microwires.Connexin43, the major gap junction protein of astrocytes, is down-regulated in inflamed white matter in an animal model of multiple sclerosis.Poly(lactic-co-glycolic) acid microspheres encapsulated in Pluronic F-127 prolong hirudin delivery and improve functional recovery from a demyelination lesion.Experimental traumatic brain injuryImmature astrocytes promote CNS axonal regeneration when combined with chondroitinase ABCCorticospinal sprouting differs according to spinal injury location and cortical origin in macaque monkeys.Enhanced functional recovery in MRL/MpJ mice after spinal cord dorsal hemisection.Role of microglia in neurotrauma.Effects of caspase-1 knockout on chronic neural recording quality and longevity: insight into cellular and molecular mechanisms of the reactive tissue response.The role of the microglia in acute CNS injury.Chondroitinase and growth factors enhance activation and oligodendrocyte differentiation of endogenous neural precursor cells after spinal cord injury.
P2860
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P2860
Cellular and molecular mechanisms of glial scarring and progressive cavitation: in vivo and in vitro analysis of inflammation-induced secondary injury after CNS trauma.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
Cellular and molecular mechani ...... ndary injury after CNS trauma.
@en
Cellular and molecular mechani ...... ndary injury after CNS trauma.
@nl
type
label
Cellular and molecular mechani ...... ndary injury after CNS trauma.
@en
Cellular and molecular mechani ...... ndary injury after CNS trauma.
@nl
prefLabel
Cellular and molecular mechani ...... ndary injury after CNS trauma.
@en
Cellular and molecular mechani ...... ndary injury after CNS trauma.
@nl
P2093
P1476
Cellular and molecular mechani ...... ondary injury after CNS trauma
@en
P2093
P304
P356
10.1523/JNEUROSCI.19-19-08182.1999
P407
P577
1999-10-01T00:00:00Z