Matrix metalloproteinase-2 and metalloproteinase-9 activities are associated with blood-brain barrier dysfunction in an animal model of severe sepsis.
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Mechanisms of long-term cognitive dysfunction of sepsis: from blood-borne leukocytes to glial cellsSac-1004, a vascular leakage blocker, reduces cerebral ischemia-reperfusion injury by suppressing blood-brain barrier disruption and inflammation.Homocysteine Induced Cerebrovascular Dysfunction: A Link to Alzheimer's Disease EtiologyCD40-CD40 Ligand Pathway is a Major Component of Acute Neuroinflammation and Contributes to Long-term Cognitive Dysfunction after Sepsis.Evaluation of New Diagnostic Biomarkers in Pediatric Sepsis: Matrix Metalloproteinase-9, Tissue Inhibitor of Metalloproteinase-1, Mid-Regional Pro-Atrial Natriuretic Peptide, and Adipocyte Fatty-Acid Binding ProteinRelationships between adiponectin and matrix metalloproteinase-9 (MMP-9) serum levels and postoperative cognitive dysfunction in elderly patients after general anesthesia.Blood-brain barrier and intestinal epithelial barrier alterations in autism spectrum disorders.Septic encephalopathy: does inflammation drive the brain crazy?TGF-beta1 regulates human brain pericyte inflammatory processes involved in neurovasculature function.Neurological consequences of systemic inflammation in the premature neonateBrain Barrier Breakdown as a Cause and Consequence of Neuroinflammation in Sepsis.Vasopressin Impairment During Sepsis Is Associated with Hypothalamic Intrinsic Apoptotic Pathway and Microglial Activation.Disrupted Tryptophan Metabolism Induced Cognitive Impairment in a Mouse Model of Sepsis-associated Encephalopathy.Endotoxin-induced lung alveolar cell injury causes brain cell damage.A Role for the Cavin-3/Matrix Metalloproteinase-9 Signaling Axis in the Regulation of PMA-Activated Human HT1080 Fibrosarcoma Cell Neoplastic Phenotype.Acute brain inflammation and oxidative damage are related to long-term cognitive deficits and markers of neurodegeneration in sepsis-survivor rats.Brain Oxidative Stress During Experimental Sepsis Is Attenuated by Simvastatin Administration.Effects of organoselenium compounds on early and late brain biochemical alterations in sepsis-survivor rats.Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment.Vitamin B6 Reduces Neurochemical and Long-Term Cognitive Alterations After Polymicrobial Sepsis: Involvement of the Kynurenine Pathway Modulation.Increased on oxidative brain injury in the diabetic rats following sepsis.Inhibition of matrix metalloproteinases-2 and -9 prevents cognitive impairment induced by pneumococcal meningitis in Wistar rats.Systemic Inflammation as a Driver of Brain Injury: the Astrocyte as an Emerging Player.Matrix Metalloproteinase-9 and Tissue Inhibitor of Matrix Metalloproteinase-1 in Sepsis after Major Abdominal Surgery.
P2860
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P2860
Matrix metalloproteinase-2 and metalloproteinase-9 activities are associated with blood-brain barrier dysfunction in an animal model of severe sepsis.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
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2013年學術文章
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2013年學術文章
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name
Matrix metalloproteinase-2 and ...... animal model of severe sepsis.
@en
Matrix metalloproteinase-2 and ...... animal model of severe sepsis.
@nl
type
label
Matrix metalloproteinase-2 and ...... animal model of severe sepsis.
@en
Matrix metalloproteinase-2 and ...... animal model of severe sepsis.
@nl
prefLabel
Matrix metalloproteinase-2 and ...... animal model of severe sepsis.
@en
Matrix metalloproteinase-2 and ...... animal model of severe sepsis.
@nl
P2093
P2860
P50
P1476
Matrix metalloproteinase-2 and ...... animal model of severe sepsis
@en
P2093
Clarissa M Comim
Cristiane Ritter
Daniel Pens Gelain
Emilia Marcelina dos Santos
Fabrícia Petronilho
Francieli Vuolo
Gustavo Feier
José Cláudio Fonseca Moreira
Matheus Pasquali
P2860
P2888
P356
10.1007/S12035-013-8433-7
P577
2013-03-12T00:00:00Z