Angiogenesis during pregnancy: all routes lead to MAPKs.

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Angiogenesis during pregnancy: all routes lead to MAPKs.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

@wuu

2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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Angiogenesis during pregnancy: all routes lead to MAPKs.

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Angiogenesis during pregnancy: all routes lead to MAPKs.

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Item

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Angiogenesis during pregnancy: all routes lead to MAPKs.

@en

Angiogenesis during pregnancy: all routes lead to MAPKs.

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Angiogenesis during pregnancy: all routes lead to MAPKs.

@en

Angiogenesis during pregnancy: all routes lead to MAPKs.

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P1476

Angiogenesis during pregnancy: all routes lead to MAPKs

@en

P2093

Xiang-Qun Hu

http://www.w3.org/2001/XMLSchema#string

P2860

Estradiol-17beta and its cytochrome P450- and catechol-O-methyltransferase-derived metabolites stimulate proliferation in uterine artery endothelial cells: role of estrogen receptor-alpha versus estrogen receptor-beta.

Deficiency in catechol-O-methyltransferase and 2-methoxyoestradiol is associated with pre-eclampsia.

A novel role for an endothelial adrenergic receptor system in mediating catecholestradiol-induced proliferation of uterine artery endothelial cells.

Aberrant synthesis, metabolism, and plasma accumulation of circulating estrogens and estrogen metabolites in preeclampsia implications for vascular dysfunction.

Convergent ERK1/2, p38 and JNK mitogen activated protein kinases (MAPKs) signalling mediate catecholoestradiol-induced proliferation of ovine uterine artery endothelial cells.

P304

4571-4572

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P31

scholarly article

P356

10.1113/JP274489

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

595

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P50

Lubo Zhang

P577

2017-06-08T00:00:00Z

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P698

28513857

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P932

5509881

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