Notch Signaling Participates in TGF-β-Induced SOST Expression Under Intermittent Compressive Stress.

about

Notch Signaling Participates in TGF-β-Induced SOST Expression Under Intermittent Compressive Stress.

Item

http://www.wikidata.org/entity/Q48157516

description

2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年学术文章

@wuu

2016年学术文章

@zh-cn

2016年学术文章

@zh-hans

2016年学术文章

@zh-my

2016年学术文章

@zh-sg

2016年學術文章

@yue

2016年學術文章

@zh

2016年學術文章

@zh-hant

name

Notch Signaling Participates i ...... termittent Compressive Stress.

@en

Notch Signaling Participates i ...... termittent Compressive Stress.

@nl

type

Item

label

Notch Signaling Participates i ...... termittent Compressive Stress.

@en

Notch Signaling Participates i ...... termittent Compressive Stress.

@nl

prefLabel

Notch Signaling Participates i ...... termittent Compressive Stress.

@en

Notch Signaling Participates i ...... termittent Compressive Stress.

@nl

P1476

Notch Signaling Participates i ...... termittent Compressive Stress.

@en

P2093

Jeeranan Manokawinchoke

http://www.w3.org/2001/XMLSchema#string

Piyamas Sumrejkanchanakij

http://www.w3.org/2001/XMLSchema#string

Prasit Pavasant

http://www.w3.org/2001/XMLSchema#string

P2860

Cyclic stretch stimulates vascular smooth muscle cell alignment by redox-dependent activation of Notch3

SOST is a ligand for LRP5/LRP6 and a Wnt signaling inhibitor

Cross-talk between the Notch and TGF-beta signaling pathways mediated by interaction of the Notch intracellular domain with Smad3

The canonical Notch signaling pathway: unfolding the activation mechanism.

Periostin associates with Notch1 precursor to maintain Notch1 expression under a stress condition in mouse cells

Cyclic strain inhibits Notch receptor signaling in vascular smooth muscle cells in vitro.

Notch and TGF-β pathways cooperatively regulate receptor protein tyrosine phosphatase-κ (PTPRK) gene expression in human primary keratinocytes.

TGF-β regulates sclerostin expression via the ECR5 enhancer.

Removal of SOST or blocking its product sclerostin rescues defects in the periodontitis mouse model.

Notch signaling promotes osteoclast maturation and resorptive activity.

P304

2221-2230

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1002/JCP.25740

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

232

http://www.w3.org/2001/XMLSchema#string

P50

Thanaphum Osathanon

P577

2016-12-14T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

27966788

http://www.w3.org/2001/XMLSchema#string