Microglia inflict delayed brain injury after subarachnoid hemorrhage. - Wikidata - awgldk - TriplyDB

Microglia inflict delayed brain injury after subarachnoid hemorrhage.

Microglia inflict delayed brain injury after subarachnoid hemorrhage.

http://www.wikidata.org/entity/Q48186929

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Aneurysmal Subarachnoid Hemorrhage and Neuroinflammation: A Comprehensive Review Soluble Toll-Like Receptors 2 and 4 in Cerebrospinal Fluid of Patients with Acute Hydrocephalus following Aneurysmal Subarachnoid Haemorrhage.The Sur1-Trpm4 channel regulates NOS2 transcription in TLR4-activated microglia.Estrogen activation of microglia underlies the sexually dimorphic differences in Nf1 optic glioma-induced retinal pathology.X-linked inhibitor of apoptosis inhibits apoptosis and preserves the blood-brain barrier after experimental subarachnoid hemorrhage.An introduction to the pathophysiology of aneurysmal subarachnoid hemorrhage.Lessons Learned about Neurodegeneration from Microglia and Monocyte Depletion Studies.Xenon Reduces Neuronal Hippocampal Damage and Alters the Pattern of Microglial Activation after Experimental Subarachnoid Hemorrhage: A Randomized Controlled Animal Trial.Detection of Neuroinflammation in a Rat Model of Subarachnoid Hemorrhage Using [18F]DPA-714 PET Imaging.The Potential Therapeutic Effects of Artesunate on Stroke and Other Central Nervous System Diseases.Time Courses of Inflammatory Markers after Aneurysmal Subarachnoid Hemorrhage and Their Possible Relevance for Future Studies.Intravascular Inflammation Triggers Intracerebral Activated Microglia and Contributes to Secondary Brain Injury After Experimental Subarachnoid Hemorrhage (eSAH).Delayed Cerebral Ischemia after Subarachnoid Hemorrhage: Beyond Vasospasm and Towards a Multifactorial Pathophysiology.Possible Role of Inflammation and Galectin-3 in Brain Injury after Subarachnoid Hemorrhage.A Systematic and Meta-Analysis of Mortality in Experimental Mouse Models Analyzing Delayed Cerebral Ischemia After Subarachnoid Hemorrhage.Microglial Lectins in Health and Neurological Diseases.Aneurysmal Subarachnoid Hemorrhage.Unique Contribution of Haptoglobin and Haptoglobin Genotype in Aneurysmal Subarachnoid Hemorrhage.

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Microglia inflict delayed brain injury after subarachnoid hemorrhage.

http://www.wikidata.org/entity/Q48186929

description

2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年學術文章

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2015年學術文章

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2015年學術文章

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name

Microglia inflict delayed brain injury after subarachnoid hemorrhage.

@en

Microglia inflict delayed brain injury after subarachnoid hemorrhage.

@nl

type

label

Microglia inflict delayed brain injury after subarachnoid hemorrhage.

@en

Microglia inflict delayed brain injury after subarachnoid hemorrhage.

@nl

prefLabel

Microglia inflict delayed brain injury after subarachnoid hemorrhage.

@en

Microglia inflict delayed brain injury after subarachnoid hemorrhage.

@nl

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S00401-015-1440-1

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Acta Neuropathologica

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Microglia inflict delayed brain injury after subarachnoid hemorrhage

@en

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Angelika Gutenberg

http://www.w3.org/2001/XMLSchema#string

Anja-Maria Davids

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Anna Elke

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Annett Müller

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Claire Gehlhaar

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Etienne Atangana

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Kati Turkowski

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Salima Magrini

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Susan Brandenburg

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Tobias Finger

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P2860

Functional impairment of microglia coincides with Beta-amyloid deposition in mice with Alzheimer-like pathology

Cerebral vasospasm after subarachnoid hemorrhage: putative role of inflammation.

Interleukin-6 deficiency reduces the brain inflammatory response and increases oxidative stress and neurodegeneration after kainic acid-induced seizures.

Cortical spreading ischaemia is a novel process involved in ischaemic damage in patients with aneurysmal subarachnoid haemorrhage

Microglia promote learning-dependent synapse formation through brain-derived neurotrophic factor.

CSF neutrophils are implicated in the development of vasospasm in subarachnoid hemorrhage.

Repeated mild traumatic brain injury causes chronic neuroinflammation, changes in hippocampal synaptic plasticity, and associated cognitive deficits.

Recurrent spreading depolarizations after subarachnoid hemorrhage decreases oxygen availability in human cerebral cortex.

The myeloid cells of the central nervous system parenchyma.

Microglia emerge from erythromyeloid precursors via Pu.1- and Irf8-dependent pathways.

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10.1007/S00401-015-1440-1

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2

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130

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Frank L. Heppner

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2015-05-09T00:00:00Z

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276067256

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