Carbon dioxide-mediated vasomotion of extra-cranial cerebral arteries in humans: a role for prostaglandins?
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Human cerebral blood flow control during hypoxia: focus on chronic pulmonary obstructive disease (COPD) and obstructive sleep apnea (OSA).Adenosine receptor-dependent signaling is not obligatory for normobaric and hypobaric hypoxia-induced cerebral vasodilation in humans.Reply from Ryan L. Hoiland and Philip N. Ainslie.Indomethacin on extra-cranial cerebral arterial vasomotion: beyond cyclooxygenase-prostaglandin inhibition.β1-Blockade increases maximal apnea duration in elite breath-hold divers.
P2860
Carbon dioxide-mediated vasomotion of extra-cranial cerebral arteries in humans: a role for prostaglandins?
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2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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2016年学术文章
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name
Carbon dioxide-mediated vasomo ...... ns: a role for prostaglandins?
@en
Carbon dioxide-mediated vasomo ...... ns: a role for prostaglandins?
@nl
type
label
Carbon dioxide-mediated vasomo ...... ns: a role for prostaglandins?
@en
Carbon dioxide-mediated vasomo ...... ns: a role for prostaglandins?
@nl
prefLabel
Carbon dioxide-mediated vasomo ...... ns: a role for prostaglandins?
@en
Carbon dioxide-mediated vasomo ...... ns: a role for prostaglandins?
@nl
P2093
P2860
P356
P1476
Carbon dioxide-mediated vasomo ...... ns: a role for prostaglandins?
@en
P2093
Anthony R Bain
Brad Monteleone
Kevin W Wildfong
Michael M Tymko
Philip N Ainslie
P2860
P304
P356
10.1113/JP272012
P407
P50
P577
2016-02-16T00:00:00Z