Malformations of hypothalamic nuclei in hyperinsulinemic offspring of rats with gestational diabetes.
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Metabolic imprinting: critical impact of the perinatal environment on the regulation of energy homeostasisThe Role of Maternal Dietary Proteins in Development of Metabolic Syndrome in OffspringReduced melanocortin production causes sexual dysfunction in male mice with POMC neuronal insulin and leptin insensitivity.Insulin and Leptin Signaling Interact in the Mouse Kiss1 Neuron during the Peripubertal Period.Low taurine, gamma-aminobutyric acid and carnosine levels in plasma of diabetic pregnant rats: consequences for the offspring.Neonatal insulin action impairs hypothalamic neurocircuit formation in response to maternal high-fat feeding.Perinatal environment and its influences on metabolic programming of offspring.Interaction of perinatal and pre-pubertal factors with genetic predisposition in the development of neural pathways involved in the regulation of energy homeostasisDevelopmental gene x environment interactions affecting systems regulating energy homeostasis and obesity.The obesity epidemic: metabolic imprinting on genetically susceptible neural circuits.Sexual dimorphism in offspring glucose-sensitive hypothalamic gene expression and physiological responses to maternal high-fat diet feeding.Maternal influence, not diabetic intrauterine environment, predicts children's energy intakeChronic sleep restriction during pregnancy--repercussion on cardiovascular and renal functioning of male offspring.Gene-environment interactions controlling energy and glucose homeostasis and the developmental origins of obesityTaurine and taurine-deficiency in the perinatal period.Higher cord C-peptide concentrations are associated with slower growth rate in the 1st year of life in girls but not in boysAcquired alterations of hypothalamic gene expression of insulin and leptin receptors and glucose transporters in prenatally high-glucose exposed three-week old chickens do not coincide with aberrant promoter DNA methylationMaternal diabetes compromises the organization of hypothalamic feeding circuits and impairs leptin sensitivity in offspring.'Fetal programming' and 'functional teratogenesis': on epigenetic mechanisms and prevention of perinatally acquired lasting health risks.Feed your head: neurodevelopmental control of feeding and metabolismSevere maternal hyperglycemia exacerbates the development of insulin resistance and fatty liver in the offspring on high fat diet.Perinatal programming of obesity.Gestational Diabetes and the Offspring: Implications in the Development of the Cardiorenal Metabolic Syndrome in OffspringEarly origins of obesity: programming the appetite regulatory system.Alterations in perivascular innervation function in mesenteric arteries from offspring of diabetic rats.Assessment of Attention Deficits in Adolescent Offspring Exposed to Maternal Type 1 Diabetes.Programming of the appetite-regulating neural network: a link between maternal overnutrition and the programming of obesity?Organizational actions of metabolic hormones.Gestational diabetes and subsequent growth patterns of offspring: the National Collaborative Perinatal Project.Maternal nutrition and the programming of obesity: The brain.A matter of insulin: developmental programming of body weight regulation.Sleep restriction during pregnancy and its effects on blood pressure and renal function among female offspring.Autonomic and Renal Alterations in the Offspring of Sleep-Restricted Mothers During Late PregnancyAnimal models for clinical and gestational diabetes: maternal and fetal outcomes.Mechanisms affecting neuroendocrine and epigenetic regulation of body weight and onset of puberty: potential implications in the child born small for gestational age (SGA).Mechanisms underlying the developmental origins of disease.Perinatal overnutrition and the programming of food preferences: pathways and mechanisms.Metabolic Syndrome in Children and Adolescents.Maternal obesity alters adiposity and monoamine function in genetically predisposed offspring.Maternal hyperglycemia leads to gender-dependent deficits in learning and memory in offspring.
P2860
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P2860
Malformations of hypothalamic nuclei in hyperinsulinemic offspring of rats with gestational diabetes.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh-hant
name
Malformations of hypothalamic ...... ats with gestational diabetes.
@en
Malformations of hypothalamic ...... ats with gestational diabetes.
@nl
type
label
Malformations of hypothalamic ...... ats with gestational diabetes.
@en
Malformations of hypothalamic ...... ats with gestational diabetes.
@nl
prefLabel
Malformations of hypothalamic ...... ats with gestational diabetes.
@en
Malformations of hypothalamic ...... ats with gestational diabetes.
@nl
P2093
P356
P1476
Malformations of hypothalamic ...... ats with gestational diabetes.
@en
P2093
A Plagemann
P356
10.1159/000017367
P577
1999-01-01T00:00:00Z