Protection from autoimmune brain inflammation in mice lacking IFN-regulatory factor-1 is associated with Th2-type cytokines.
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CAWS administration increases the expression of interferon γ and complement factors that lead to severe vasculitis in DBA/2 miceSystems-based medicine approaches to understand and treat complex diseases. The example of multiple sclerosis.The macrophage IRF8/IRF1 regulome is required for protection against infections and is associated with chronic inflammationOverexpression of the dominant-negative form of interferon regulatory factor 1 in oligodendrocytes protects against experimental autoimmune encephalomyelitisInterleukin 10 protects the brain microcirculation from spirochetal injuryAstrocytes play a key role in EAE pathophysiology by orchestrating in the CNS the inflammatory response of resident and peripheral immune cells and by suppressing remyelination.
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Protection from autoimmune brain inflammation in mice lacking IFN-regulatory factor-1 is associated with Th2-type cytokines.
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2003 nî lūn-bûn
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name
Protection from autoimmune bra ...... iated with Th2-type cytokines.
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Protection from autoimmune bra ...... iated with Th2-type cytokines.
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type
label
Protection from autoimmune bra ...... iated with Th2-type cytokines.
@en
Protection from autoimmune bra ...... iated with Th2-type cytokines.
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prefLabel
Protection from autoimmune bra ...... iated with Th2-type cytokines.
@en
Protection from autoimmune bra ...... iated with Th2-type cytokines.
@nl
P2860
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Protection from autoimmune bra ...... iated with Th2-type cytokines.
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Claudia Uthoff-Hachenberg
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P304
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10.1093/INTIMM/DXG086
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2003-07-01T00:00:00Z