Novel therapeutic concepts for inflammatory bowel disease-from bench to bedside.

about

Novel therapeutic concepts for inflammatory bowel disease-from bench to bedside.

Item

http://www.wikidata.org/entity/Q48286065

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Novel therapeutic concepts for inflammatory bowel disease-from bench to bedside.

@en

Novel therapeutic concepts for inflammatory bowel disease-from bench to bedside.

@nl

type

Item

label

Novel therapeutic concepts for inflammatory bowel disease-from bench to bedside.

@en

Novel therapeutic concepts for inflammatory bowel disease-from bench to bedside.

@nl

prefLabel

Novel therapeutic concepts for inflammatory bowel disease-from bench to bedside.

@en

Novel therapeutic concepts for inflammatory bowel disease-from bench to bedside.

@nl

P1476

Novel therapeutic concepts for inflammatory bowel disease-from bench to bedside

@en

P2093

Holger K Eltzschig

http://www.w3.org/2001/XMLSchema#string

Sean P Colgan

http://www.w3.org/2001/XMLSchema#string

P2860

General involvement of hypoxia-inducible factor 1 in transcriptional response to hypoxia

Crucial role for ecto-5'-nucleotidase (CD73) in vascular leakage during hypoxia.

Muc2-deficient mice spontaneously develop colitis, indicating that MUC2 is critical for colonic protection

CD39 is incorporated into plasma microparticles where it maintains functional properties and impacts endothelial activation.

Crosstalk between Microbiota-Derived Short-Chain Fatty Acids and Intestinal Epithelial HIF Augments Tissue Barrier Function.

MicroRNA214 Is Associated With Progression of Ulcerative Colitis, and Inhibition Reduces Development of Colitis and Colitis-Associated Cancer in Mice

Epithelial-specific A2B adenosine receptor signaling protects the colonic epithelial barrier during acute colitis

Ecto-nucleoside triphosphate diphosphohydrolase 7 controls Th17 cell responses through regulation of luminal ATP in the small intestine

Adenosine and gastrointestinal inflammation.

From the Cover: CD39 deletion exacerbates experimental murine colitis and human polymorphisms increase susceptibility to inflammatory bowel disease.

P2888

s00109-017-1574-9

P304

899-903

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

editorial

P356

10.1007/S00109-017-1574-9

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Viola Neudecker

P577

2017-09-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28780611

http://www.w3.org/2001/XMLSchema#string

P932

5800411

http://www.w3.org/2001/XMLSchema#string