Progressive neuropathology and cognitive decline in a single Arctic APP transgenic mouse model.
about
Lesion of the subiculum reduces the spread of amyloid beta pathology to interconnected brain regions in a mouse model of Alzheimer's diseaseBehavioral assays with mouse models of Alzheimer's disease: practical considerations and guidelines.Mouse brain magnetic resonance microscopy: Applications in Alzheimer disease.Serial propagation of distinct strains of Aβ prions from Alzheimer's disease patients.Accelerating drug discovery for Alzheimer's disease: best practices for preclinical animal studiesOctodon degus: a strong attractor for Alzheimer research.Amyloid-beta: a crucial factor in Alzheimer's disease.Observations in APP bitransgenic mice suggest that diffuse and compact plaques form via independent processes in Alzheimer's disease.Vascular β-amyloid and early astrocyte alterations impair cerebrovascular function and cerebral metabolism in transgenic arcAβ mice.Modulation of the endoplasmic reticulum-mitochondria interface in Alzheimer's disease and related models.A longitudinal study of behavioral deficits in an AβPP transgenic mouse model of Alzheimer's disease.Insights into Alzheimer disease pathogenesis from studies in transgenic animal models.Imaging of cerebrovascular pathology in animal models of Alzheimer's diseaseAggregation of the Inflammatory S100A8 Precedes Aβ Plaque Formation in Transgenic APP Mice: Positive Feedback for S100A8 and Aβ Productions.Mitochondrial dysfunction in a transgenic mouse model expressing human amyloid precursor protein (APP) with the Arctic mutation.Mouse models of neurodegenerative disease: preclinical imaging and neurovascular component.Critical role of somatostatin receptor 2 in the vulnerability of the central noradrenergic system: new aspects on Alzheimer's disease.Reduced nitric oxide bioavailability mediates cerebroarterial dysfunction independent of cerebral amyloid angiopathy in a mouse model of Alzheimer's disease.Trans-Synaptic Spread of Amyloid-β in Alzheimer's Disease: Paths to β-Amyloidosis.Arctic mutant Aβ40 aggregates on α7 nicotinic acetylcholine receptors and inhibits their functions.
P2860
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P2860
Progressive neuropathology and cognitive decline in a single Arctic APP transgenic mouse model.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Progressive neuropathology and ...... ic APP transgenic mouse model.
@en
Progressive neuropathology and ...... ic APP transgenic mouse model.
@nl
type
label
Progressive neuropathology and ...... ic APP transgenic mouse model.
@en
Progressive neuropathology and ...... ic APP transgenic mouse model.
@nl
prefLabel
Progressive neuropathology and ...... ic APP transgenic mouse model.
@en
Progressive neuropathology and ...... ic APP transgenic mouse model.
@nl
P2093
P50
P1476
Progressive neuropathology and ...... ic APP transgenic mouse model.
@en
P2093
Jan Näslund
Karin Dillner
Lena Lilius
Mikio Aoki
Shunwei Zhu
P304
P356
10.1016/J.NEUROBIOLAGING.2009.02.021
P577
2009-03-28T00:00:00Z