Transgenic overexpression of interleukin (IL)-10 in the lung causes mucus metaplasia, tissue inflammation, and airway remodeling via IL-13-dependent and -independent pathways. - Wikidata - awgldk - TriplyDB

Transgenic overexpression of interleukin (IL)-10 in the lung causes mucus metaplasia, tissue inflammation, and airway remodeling via IL-13-dependent and -independent pathways.

Transgenic overexpression of interleukin (IL)-10 in the lung causes mucus metaplasia, tissue inflammation, and airway remodeling via IL-13-dependent and -independent pathways.

http://www.wikidata.org/entity/Q48294439

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The role of pro- and anti-inflammatory responses in silica-induced lung fibrosis.Scleroderma pathogenesis: a pivotal role for fibroblasts as effector cells Uncoupling between inflammatory and fibrotic responses to silica: evidence from MyD88 knockout mice Gene expression profiles reveal increased mClca3 (Gob5) expression and mucin production in a murine model of asbestos-induced fibrogenesis Accelerated and progressive and lethal liver fibrosis in mice that lack interleukin (IL)-10, IL-12p40, and IL-13Rα2.Acellular pertussis vaccine protects against exacerbation of allergic asthma due to Bordetella pertussis in a murine model.Reversing the defective induction of IL-10-secreting regulatory T cells in glucocorticoid-resistant asthma patients Adenosine metabolism and murine strain-specific IL-4-induced inflammation, emphysema, and fibrosis.Latent infection by γherpesvirus stimulates profibrotic mediator release from multiple cell types.New concepts of IL-10-induced lung fibrosis: fibrocyte recruitment and M2 activation in a CCL2/CCR2 axis Protective effects of basic fibroblast growth factor in the development of emphysema induced by interferon-γ The role of macrophage IL-10/innate IFN interplay during virus-induced asthma.Epithelial cell modulation of airway fibrosis in asthma.Making More MUCS.Interactions between tissue fibroblasts in lymph nodes and Hodgkin/Reed-Sternberg cells.IL-13 Stimulates Proliferation and Expression of Mucin and Immunomodulatory Genes in Cultured Conjunctival Goblet Cells Fibrotic disease and the T(H)1/T(H)2 paradigm.IL-13Ralpha2 and IL-10 coordinately suppress airway inflammation, airway-hyperreactivity, and fibrosis in mice Airway remodeling in asthma: therapeutic implications of mechanisms.IL-18 induces emphysema and airway and vascular remodeling via IFN-γ, IL-17A, and IL-13 Aerobic Exercise Attenuated Bleomycin-Induced Lung Fibrosis in Th2-Dominant Mice.Vascular endothelial growth factor (VEGF) induces remodeling and enhances TH2-mediated sensitization and inflammation in the lung.Pathogenesis of respiratory syncytial virus infection in the murine model New drugs targeting Th2 lymphocytes in asthma.Mucin overproduction in chronic inflammatory lung disease.IL-10 Enhances IgE-Mediated Mast Cell Responses and Is Essential for the Development of Experimental Food Allergy in IL-10-Deficient Mice.Esophageal remodeling develops as a consequence of tissue specific IL-5-induced eosinophilia.Reversal of allergen-induced airway remodeling by CysLT1 receptor blockade.Math1, retinoic acid, and TNF-α synergistically promote the differentiation of mucous cells in mouse middle ear epithelial cells in vitro.Tissue inhibitor of metalloproteinase-1 modulates allergic lung inflammation in murine asthma.Host immune responses to chlamydial inclusion membrane proteins B and C in Chlamydia trachomatis infected women with or without fertility disorders Role of breast regression protein 39 (BRP-39)/chitinase 3-like-1 in Th2 and IL-13-induced tissue responses and apoptosis.Interleukin-10 prevents diet-induced insulin resistance by attenuating macrophage and cytokine response in skeletal muscle.Antagonism of Interleukin-17A ameliorates experimental hepatic fibrosis by restoring the IL-10/STAT3-suppressed autophagy in hepatocytes.Transgenic modelling of cytokine polarization in the lung.Mucin production and mucous cell metaplasia in otitis media.Beneficial Effects of Prebiotic Saccharomyces cerevisiae Mannan on Allergic Asthma Mouse Models.Adenosine mediates IL-13-induced inflammation and remodeling in the lung and interacts in an IL-13-adenosine amplification pathway Pathobiology of cigarette smoke-induced chronic obstructive pulmonary disease.IL-10 enhances CCL2 release and chemotaxis induced by CCL16 in human monocytes.

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P2860

Transgenic overexpression of interleukin (IL)-10 in the lung causes mucus metaplasia, tissue inflammation, and airway remodeling via IL-13-dependent and -independent pathways.

http://www.wikidata.org/entity/Q48294439

description

2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年學術文章

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2002年學術文章

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2002年學術文章

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name

Transgenic overexpression of i ...... ent and -independent pathways.

@en

Transgenic overexpression of i ...... ent and -independent pathways.

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type

label

Transgenic overexpression of i ...... ent and -independent pathways.

@en

Transgenic overexpression of i ...... ent and -independent pathways.

@nl

prefLabel

Transgenic overexpression of i ...... ent and -independent pathways.

@en

Transgenic overexpression of i ...... ent and -independent pathways.

@nl

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Transgenic overexpression of i ...... ent and -independent pathways.

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P2093

Holger Link

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Jack A Elias

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Joseph E Craft

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Lauren Cohn

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Sungsoo Jung

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Teresa R Johnson

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P2860

Complete sequence of the human mucin MUC4: a putative cell membrane-associated mucin

Interleukin-13: central mediator of allergic asthma

A CD4+ T-cell subset inhibits antigen-specific T-cell responses and prevents colitis

Recombinant human interleukin 10 in the treatment of patients with mild to moderately active Crohn's disease. The Interleukin 10 Inflammatory Bowel Disease Cooperative Study Group.

Allergen-induced IL-9 directly stimulates mucin transcription in respiratory epithelial cells.

Interleukin-10 inhibits antigen-induced cellular recruitment into the airways of sensitized mice.

Inducible targeting of IL-13 to the adult lung causes matrix metalloproteinase- and cathepsin-dependent emphysema

IL-10 is necessary for the expression of airway hyperresponsiveness but not pulmonary inflammation after allergic sensitization

Role of gob-5 in mucus overproduction and airway hyperresponsiveness in asthma.

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10.1074/JBC.M206395200

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38

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277

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Robert J. Homer

Barney S. Graham

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2002-07-09T00:00:00Z

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11271632

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