Activation of microglial N-methyl-D-aspartate receptors triggers inflammation and neuronal cell death in the developing and mature brain. - Wikidata - awgldk - TriplyDB

Activation of microglial N-methyl-D-aspartate receptors triggers inflammation and neuronal cell death in the developing and mature brain.

Activation of microglial N-methyl-D-aspartate receptors triggers inflammation and neuronal cell death in the developing and mature brain.

http://www.wikidata.org/entity/Q48316394

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Neuroinflammation and psychiatric illness Treatment of Complex Regional Pain Syndrome (CRPS) using low dose naltrexone (LDN)Bridge between neuroimmunity and traumatic brain injury Microglia protect against brain injury and their selective elimination dysregulates neuronal network activity after stroke.Neuronal Hyperactivity Disturbs ATP Microgradients, Impairs Microglial Motility, and Reduces Phagocytic Receptor Expression Triggering Apoptosis/Microglial Phagocytosis Uncoupling Development of NMDAR antagonists with reduced neurotoxic side effects: a study on GK11 Inflammation and the neural diathesis-stress hypothesis of schizophrenia: a reconceptualization NR2B phosphorylation at tyrosine 1472 contributes to brain injury in a rodent model of neonatal hypoxia-ischemia Neuronal hyperactivity recruits microglial processes via neuronal NMDA receptors and microglial P2Y12 receptors after status epilepticus.Developmental expression of N-methyl-D-aspartate (NMDA) receptor subunits in human white and gray matter: potential mechanism of increased vulnerability in the immature brain RNA-sequencing reveals oligodendrocyte and neuronal transcripts in microglia relevant to central nervous system disease.Radix Scrophulariae extracts (harpagoside) suppresses hypoxia-induced microglial activation and neurotoxicity.Effects of long-term treatment on brain volume in patients with obstructive sleep apnea syndrome Characterization of phenotype markers and neuronotoxic potential of polarised primary microglia in vitro Perinatal brain damage: The term infant.CD163 promotes hematoma absorption and improves neurological functions in patients with intracerebral hemorrhage.PET Imaging with [(18)F]FSPG Evidences the Role of System xc(-) on Brain Inflammation Following Cerebral Ischemia in Rats.Systemic and localized extra-central nervous system bacterial infections and the risk of dementia among US veterans: A retrospective cohort study.The Anti-Inflammatory Effects of the Small Molecule Pifithrin-µ on BV2 Microglia.Bone fracture exacerbates murine ischemic cerebral injury.Myeloid C/EBPβ deficiency reshapes microglial gene expression and is protective in experimental autoimmune encephalomyelitis.Microglial control of neuronal activity.Immune dysregulation and autoimmunity in bipolar disorder: Synthesis of the evidence and its clinical application.Microglia toxicity in preterm brain injury.Glia and zinc in ageing and Alzheimer's disease: a mechanism for cognitive decline?Inflammation and epilepsy in the developing brain: clinical and experimental evidence.NADPH oxidases in oxidant production by microglia: activating receptors, pharmacology and association with disease.Ethanol and Other Short-Chain Alcohols Inhibit NLRP3 Inflammasome Activation through Protein Tyrosine Phosphatase Stimulation.Memantine improves outcomes after repetitive traumatic brain injury.Ionotropic glutamate receptors: Which ones, when, and where in the mammalian neocortex.Protection of Radial Glial-Like Cells in the Hippocampus of APP/PS1 Mice: a Novel Mechanism of Memantine in the Treatment of Alzheimer's Disease.Parkinson's disease: Microglial/macrophage-induced immunoexcitotoxicity as a central mechanism of neurodegeneration Synaptoimmunology - roles in health and disease.System xC- is a mediator of microglial function and its deletion slows symptoms in amyotrophic lateral sclerosis mice.Integrative genomics of microglia implicates DLG4 (PSD95) in the white matter development of preterm infants.Xenon Reduces Neuronal Hippocampal Damage and Alters the Pattern of Microglial Activation after Experimental Subarachnoid Hemorrhage: A Randomized Controlled Animal Trial.Tissue-Type Plasminogen Activator-A296-299 Prevents Impairment of Cerebral Autoregulation After Stroke Through Lipoprotein-Related Receptor-Dependent Increase in cAMP and p38.Memantine for the Treatment of Dementia: A Review on its Current and Future Applications.Contribution of mast cells to injury mechanisms in a mouse model of pediatric traumatic brain injury.Histamine H3 receptor in primary mouse microglia inhibits chemotaxis, phagocytosis, and cytokine secretion.

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P2860

Activation of microglial N-methyl-D-aspartate receptors triggers inflammation and neuronal cell death in the developing and mature brain.

http://www.wikidata.org/entity/Q48316394

description

2012 nî lūn-bûn

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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name

Activation of microglial N-met ...... e developing and mature brain.

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Activation of microglial N-met ...... e developing and mature brain.

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type

label

Activation of microglial N-met ...... e developing and mature brain.

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Activation of microglial N-met ...... e developing and mature brain.

@nl

prefLabel

Activation of microglial N-met ...... e developing and mature brain.

@en

Activation of microglial N-met ...... e developing and mature brain.

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Annals of Neurology

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Activation of microglial N-met ...... e developing and mature brain.

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Alain Lombet

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Annemieke Kavelaars

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Catherine Verney

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Elodie Gouadon

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Frédérique Rene

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Gauthier Loron

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Graham L Collingridge

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Jean Mantz

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Jean-Philippe Loeffler

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Julien Josserand

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P2860

TNF signaling inhibition in the CNS: implications for normal brain function and neurodegenerative disease

Resting microglial cells are highly dynamic surveillants of brain parenchyma in vivo

Cytokines and acute neurodegeneration

Minocycline provides neuroprotection against N-methyl-D-aspartate neurotoxicity by inhibiting microglia

PSD-95 is a negative regulator of the tyrosine kinase Src in the NMDA receptor complex

Microglia: active sensor and versatile effector cells in the normal and pathologic brain

Microglia-mediated neurotoxicity: uncovering the molecular mechanisms

Fate mapping analysis reveals that adult microglia derive from primitive macrophages

Magnesium gates glutamate-activated channels in mouse central neurones

The LTP Program: a data acquisition program for on-line analysis of long-term potentiation and other synaptic events.

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536-549

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10.1002/ANA.23626

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4

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72

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Pierre Gressens

Stéphane Peineau

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2012-10-01T00:00:00Z

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232738947

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23109148

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