about
Metabotropic glutamate receptors in the thalamocortical network: strategic targets for the treatment of absence epilepsy.Positive allosteric modulation of metabotropic glutamate 4 (mGlu4) receptors enhances spontaneous and evoked absence seizures.Protective role for type 4 metabotropic glutamate receptors against ischemic brain damageHead-to head comparison of mGlu1 and mGlu5 receptor activation in chronic treatment of absence epilepsy in WAG/Rij rats.Potentiation of mGlu5 receptors with the novel enhancer, VU0360172, reduces spontaneous absence seizures in WAG/Rij ratsProtective role for type-1 metabotropic glutamate receptors against spike and wave discharges in the WAG/Rij rat model of absence epilepsy.The anti-absence effect of mGlu5 receptor amplification with VU0360172 is maintained during and after antiepileptogenesis.Alterations in the α2 δ ligand, thrombospondin-1, in a rat model of spontaneous absence epilepsy and in patients with idiopathic/genetic generalized epilepsies.The α2δ Subunit and Absence Epilepsy: Beyond Calcium Channels?WAG/Rij rats show a reduced expression of CB₁ receptors in thalamic nuclei and respond to the CB₁ receptor agonist, R(+)WIN55,212-2, with a reduced incidence of spike-wave discharges.Abnormal Hippocampal Melatoninergic System: A Potential Link between Absence Epilepsy and Depression-Like Behavior in WAG/Rij Rats?
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description
hulumtuese
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onderzoeker
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researcher
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հետազոտող
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name
Ines Santolini
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Ines Santolini
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Ines Santolini
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Ines Santolini
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Ines Santolini
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type
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Ines Santolini
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Ines Santolini
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Ines Santolini
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Ines Santolini
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Ines Santolini
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Ines Santolini
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Ines Santolini
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Ines Santolini
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Ines Santolini
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Ines Santolini
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P1053
K-2707-2016
P106
P1153
35084302400
P21
P31
P3829
P496
0000-0002-4274-2366