Role of peroxynitrite in secondary oxidative damage after spinal cord injury. - Wikidata - awgldk - TriplyDB

Role of peroxynitrite in secondary oxidative damage after spinal cord injury.

Role of peroxynitrite in secondary oxidative damage after spinal cord injury.

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Oxidative stress, DNA damage, and the telomeric complex as therapeutic targets in acute neurodegeneration Olprinone attenuates the acute inflammatory response and apoptosis after spinal cord trauma in mice Syndromics: a bioinformatics approach for neurotrauma research Identifying the Long-Term Role of Inducible Nitric Oxide Synthase after Contusive Spinal Cord Injury Using a Transgenic Mouse Model.Beneficial effects of secretory leukocyte protease inhibitor after spinal cord injury.Therapeutic effect of apocynin through antioxidant activity and suppression of apoptosis and inflammation after spinal cord injury.Targeted siRNA delivery reduces nitric oxide mediated cell death after spinal cord injury Temporal and spatial dynamics of peroxynitrite-induced oxidative damage after spinal cord contusion injury.Pathology dynamics predict spinal cord injury therapeutic success Antioxidant therapies in traumatic brain and spinal cord injury Lipid peroxidation-derived reactive aldehydes directly and differentially impair spinal cord and brain mitochondrial function.A calpain inhibitor enhances the survival of Schwann cells in vitro and after transplantation into the injured spinal cord Location and level of Etk expression in neurons are associated with varied severity of traumatic brain injury Antiinflammatory activity of melatonin in central nervous system Antioxidant therapies for acute spinal cord injury.Novel neuroinflammatory targets in the chronically injured spinal cord Inhibition of NADPH oxidase activation in oligodendrocytes reduces cytotoxicity following trauma.Lewis, Fischer 344, and sprague-dawley rats display differences in lipid peroxidation, motor recovery, and rubrospinal tract preservation after spinal cord injury.Unmyelinated axons show selective rostrocaudal pathology in the corpus callosum after traumatic brain injury Lipid peroxidation in brain or spinal cord mitochondria after injury Dietary Supplementation with Organoselenium Accelerates Recovery of Bladder Expression, but Does Not Improve Locomotor Function, following Spinal Cord Injury.Acute molecular perturbation of inducible nitric oxide synthase with an antisense approach enhances neuronal preservation and functional recovery after contusive spinal cord injury Cellular and subcellular oxidative stress parameters following severe spinal cord injury.Correlation of Oxidative and Antioxidative Processes in the Blood of Patients with Cervical Spinal Cord Injury.Phenelzine mitochondrial functional preservation and neuroprotection after traumatic brain injury related to scavenging of the lipid peroxidation-derived aldehyde 4-hydroxy-2-nonenal.Inflammation and its role in neuroprotection, axonal regeneration and functional recovery after spinal cord injury.Na+/H+ exchanger 1 inhibition reverses manifestation of peripheral diabetic neuropathy in type 1 diabetic rats.Potential Therapeutic Targets for PPARgamma after Spinal Cord Injury.Reactions of peroxynitrite with uric acid: formation of reactive intermediates, alkylated products and triuret, and in vivo production of triuret under conditions of oxidative stress Pharmacological evidence for a role of peroxynitrite in the pathophysiology of spinal cord injury.Tempol protection of spinal cord mitochondria from peroxynitrite-induced oxidative damage.Protective effect of geraniol inhibits inflammatory response, oxidative stress and apoptosis in traumatic injury of the spinal cord through modulation of NF-κB and p38 MAPK.S-Nitrosation of β-catenin and p120 catenin: a novel regulatory mechanism in endothelial hyperpermeability.Spinal Cord Injury and Related Clinical Trials.Zonisamide-loaded triblock copolymer nanomicelles as a novel drug delivery system for the treatment of acute spinal cord injury An update on spinal cord injury research.Oxidative stress and antioxidative parameters in patients with spinal cord injury: implications in the pathogenesis of disease.Propitious Therapeutic Modulators to Prevent Blood-Spinal Cord Barrier Disruption in Spinal Cord Injury.The age factor in axonal repair after spinal cord injury: A focus on neuron-intrinsic mechanisms.NADPH oxidase isoform expression is temporally regulated and may contribute to microglial/macrophage polarization after spinal cord injury.

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Role of peroxynitrite in secondary oxidative damage after spinal cord injury.

http://www.wikidata.org/entity/Q48332415

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2006 nî lūn-bûn

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2006年の論文

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2006年学术文章

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2006年學術文章

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Role of peroxynitrite in secondary oxidative damage after spinal cord injury.

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Role of peroxynitrite in secondary oxidative damage after spinal cord injury.

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Role of peroxynitrite in secondary oxidative damage after spinal cord injury.

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Role of peroxynitrite in secondary oxidative damage after spinal cord injury.

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Role of peroxynitrite in secondary oxidative damage after spinal cord injury.

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Role of peroxynitrite in secondary oxidative damage after spinal cord injury.

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J.1471-4159.2006.04312.X

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Journal of Neurochemistry

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Role of peroxynitrite in secondary oxidative damage after spinal cord injury.

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Alexander G Rabchevsky

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Edward D Hall

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Yiqin Xiong

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P2860

Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide

Oxidant, mitochondria and calcium: an overview.

Calpain and caspase: can you tell the difference?

Uric acid protects against secondary damage after spinal cord injury.

Calpain and its involvement in the pathophysiology of CNS injuries and diseases: therapeutic potential of calpain inhibitors for prevention of neurodegeneration.

Modification of histidine residues in proteins by reaction with 4-hydroxynonenal.

Calcium fluxes, calcium antagonists, and calcium-related pathology in brain ischemia, hypoglycemia, and spreading depression: a unifying hypothesis.

Iron toxicity and oxygen radicals.

Central nervous system trauma and stroke. I. Biochemical considerations for oxygen radical formation and lipid peroxidation.

Central nervous system trauma and stroke. II. Physiological and pharmacological evidence for involvement of oxygen radicals and lipid peroxidation.

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