Lipopolysaccharide-induced inhibition of connexin43 gap junction communication in astrocytes is mediated by downregulation of caveolin-3.
about
Role of caveolin-1 and caveolae signaling in endotoxemia and sepsisDisruption in connexin-based communication is associated with intracellular Ca²⁺ signal alterations in astrocytes from Niemann-Pick type C miceReciprocal regulation between proinflammatory cytokine-induced inducible NO synthase (iNOS) and connexin43 in bladder smooth muscle cellsUpregulation of connexins 30 and 32 gap junctions in rat hippocampus at transcription level by chronic central injection of lipopolysaccharideAndrographolide induces apoptosis of C6 glioma cells via the ERK-p53-caspase 7-PARP pathway.Insulin-like growth factor-I gene delivery to astrocytes reduces their inflammatory response to lipopolysaccharideLipopolysaccharide induces degradation of connexin43 in rat astrocytes via the ubiquitin-proteasome proteolytic pathwaySex differences in the inflammatory response of primary astrocytes to lipopolysaccharideLipopolysaccharide modulates astrocytic S100B secretion: a study in cerebrospinal fluid and astrocyte cultures from ratsHYS-32-Induced Microtubule Catastrophes in Rat Astrocytes Involves the PI3K-GSK3beta Signaling PathwayRegulation of gap junction channels and hemichannels by phosphorylation and redox changes: a revision.The role of the innate immune system in Alzheimer's disease and frontotemporal lobar degeneration: an eye on microglia.Alpha 2 Na+,K+-ATPase silencing induces loss of inflammatory response and ouabain protection in glial cells.Connexins and their channels in inflammation.Connexin Channels at the Glio-Vascular Interface: Gatekeepers of the Brain.Effect of chronic intracerebroventricluar administration of lipopolysaccharide on connexin43 protein expression in rat hippocampus.Decreased connexin 43 in astrocytes inhibits the neuroinflammatory reaction in an acute mouse model of neonatal sepsis.L-type voltage-operated calcium channels contribute to astrocyte activation In vitro.Spinal astrocytes stimulated by tumor necrosis factor-α and/or interferon-γ attenuate connexin 43-gap junction via c-jun terminal kinase activity.KV 7/M channels as targets for lipopolysaccharide-induced inflammatory neuronal hyperexcitability.Reversal of rotenone-induced dysfunction of astrocytic connexin43 by opening mitochondrial ATP-sensitive potassium channels.Are Synchronized Changes in Connexin-43 and Caveolin-3 a Bystander Effect in a Phoneutria nigriventer Venom Model of Blood-Brain Barrier Breakdown?
P2860
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P2860
Lipopolysaccharide-induced inhibition of connexin43 gap junction communication in astrocytes is mediated by downregulation of caveolin-3.
description
2010 nî lūn-bûn
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年學術文章
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name
Lipopolysaccharide-induced inh ...... downregulation of caveolin-3.
@en
Lipopolysaccharide-induced inh ...... downregulation of caveolin-3.
@nl
type
label
Lipopolysaccharide-induced inh ...... downregulation of caveolin-3.
@en
Lipopolysaccharide-induced inh ...... downregulation of caveolin-3.
@nl
prefLabel
Lipopolysaccharide-induced inh ...... downregulation of caveolin-3.
@en
Lipopolysaccharide-induced inh ...... downregulation of caveolin-3.
@nl
P2093
P1476
Lipopolysaccharide-induced inh ...... downregulation of caveolin-3.
@en
P2093
Chih-Kai Liao
Hwai-Shi Wang
Jiahn-Chun Wu
Seu-Mei Wang
P304
P356
10.1016/J.BIOCEL.2010.01.016
P577
2010-01-20T00:00:00Z