Gene expression in brain during cuprizone-induced demyelination and remyelination.
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Downregulation of oligodendrocyte transcripts is associated with impaired prefrontal cortex function in rats.Tract-Based Spatial Statistics (TBSS) of diffusion tensor imaging data in alcohol dependence: abnormalities of the motivational neurocircuitry.Therapeutic inhibition of soluble brain TNF promotes remyelination by increasing myelin phagocytosis by microglia17beta-estradiol protects male mice from cuprizone-induced demyelination and oligodendrocyte loss.CNS-specific expression of C3a and C5a exacerbate demyelination severity in the cuprizone model.Myelin, copper, and the cuprizone model of schizophreniaNeurobiological effects of sphingosine 1-phosphate receptor modulation in the cuprizone model.Oligodendrocytes and progenitors become progressively depleted within chronically demyelinated lesionsMyelin-phagocytosing macrophages modulate autoreactive T cell proliferationZfp488 promotes oligodendrocyte differentiation of neural progenitor cells in adult mice after demyelination.Microglial recruitment, activation, and proliferation in response to primary demyelination.Astrogliosis during acute and chronic cuprizone demyelination and implications for remyelinationRemyelinating Oligodendrocyte Precursor Cell miRNAs from the Sfmbt2 Cluster Promote Cell Cycle Arrest and DifferentiationThe cuprizone model for demyelination.The crucial role of Erk2 in demyelinating inflammation in the central nervous system.Enhancing the ability of experimental autoimmune encephalomyelitis to serve as a more rigorous model of multiple sclerosis through refinement of the experimental designComparative prion disease gene expression profiling using the prion disease mimetic, cuprizone.Glial response during cuprizone-induced de- and remyelination in the CNS: lessons learned.The benefits and detriments of macrophages/microglia in models of multiple sclerosisReceptor for advanced glycation end products (RAGE) and its ligands: focus on spinal cord injury.Levels of BDNF impact oligodendrocyte lineage cells following a cuprizone lesion.Interleukin-13 immune gene therapy prevents CNS inflammation and demyelination via alternative activation of microglia and macrophages.Neurotoxocarosis alters myelin protein gene transcription and expression.Locomotor activity and anxiety status, but not spatial working memory, are affected in mice after brief exposure to cuprizone.Hyperpolarized 13C MR metabolic imaging can detect neuroinflammation in vivo in a multiple sclerosis murine model.An Alternative Cuprizone-Induced Demyelination and Remyelination Mouse Model.MHC class II exacerbates demyelination in vivo independently of T cells.Preliminary Observations on Sensitivity and Specificity of Magnetization Transfer Asymmetry for Imaging Myelin of Rat Brain at High Field.Alterations in metabolism and gene expression in brain regions during cuprizone-induced demyelination and remyelination.Abbreviated exposure to cuprizone is sufficient to induce demyelination and oligodendrocyte loss.Astrocytes regulate myelin clearance through recruitment of microglia during cuprizone-induced demyelination.Functional antagonism of sphingosine-1-phosphate receptor 1 prevents cuprizone-induced demyelination.Dynamics of oligodendrocyte responses to anterograde axonal (Wallerian) and terminal degeneration in normal and TNF-transgenic mice.Improvement in disability after alemtuzumab treatment of multiple sclerosis is associated with neuroprotective autoimmunity.Cuprizone Model as a Tool for Preclinical Studies of the Efficacy of Multiple Sclerosis Diagnosis and Therapy.Cuprizone-Containing Pellets Are Less Potent to Induce Consistent Demyelination in the Corpus Callosum of C57BL/6 Mice.Epimedium flavonoids ameliorate neuropathological changes and increases IGF-1 expression in C57BL/6 mice exposed to cuprizone.Episodic demyelination and subsequent remyelination within the murine central nervous system: changes in axonal calibre.Tissue transglutaminase activity is involved in the differentiation of oligodendrocyte precursor cells into myelin-forming oligodendrocytes during CNS remyelination.Sequential myelin protein expression during remyelination reveals fast and efficient repair after central nervous system demyelination.
P2860
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P2860
Gene expression in brain during cuprizone-induced demyelination and remyelination.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Gene expression in brain during cuprizone-induced demyelination and remyelination.
@en
Gene expression in brain during cuprizone-induced demyelination and remyelination.
@nl
type
label
Gene expression in brain during cuprizone-induced demyelination and remyelination.
@en
Gene expression in brain during cuprizone-induced demyelination and remyelination.
@nl
prefLabel
Gene expression in brain during cuprizone-induced demyelination and remyelination.
@en
Gene expression in brain during cuprizone-induced demyelination and remyelination.
@nl
P2093
P356
P1476
Gene expression in brain during cuprizone-induced demyelination and remyelination.
@en
P2093
Barrett CV
Hostettler JD
Matsushima GK
P304
P356
10.1006/MCNE.1998.0715
P577
1998-11-01T00:00:00Z