IL-17A Promotes Granulocyte Infiltration, Myelin Loss, Microglia Activation, and Behavioral Deficits During Cuprizone-Induced Demyelination.
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Evaluation of plasma cytokines in patients with cocaine use disorders in abstinence identifies transforming growth factor alpha (TGFα) as a potential biomarker of consumption and dual diagnosis.Combination of cuprizone and experimental autoimmune encephalomyelitis to study inflammatory brain lesion formation and progression.Acute inhalation of combustion smoke triggers neuroinflammation and persistent anxiety-like behavior in the mouse.Prenatal influenza vaccination rescues impairments of social behavior and lamination in a mouse model of autism
P2860
IL-17A Promotes Granulocyte Infiltration, Myelin Loss, Microglia Activation, and Behavioral Deficits During Cuprizone-Induced Demyelination.
description
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name
IL-17A Promotes Granulocyte In ...... prizone-Induced Demyelination.
@en
IL-17A Promotes Granulocyte In ...... prizone-Induced Demyelination.
@nl
type
label
IL-17A Promotes Granulocyte In ...... prizone-Induced Demyelination.
@en
IL-17A Promotes Granulocyte In ...... prizone-Induced Demyelination.
@nl
prefLabel
IL-17A Promotes Granulocyte In ...... prizone-Induced Demyelination.
@en
IL-17A Promotes Granulocyte In ...... prizone-Induced Demyelination.
@nl
P2093
P2860
P1476
IL-17A Promotes Granulocyte In ...... uprizone-Induced Demyelination
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P2093
Iain L Campbell
Louisa Nitsch
Marcus Müller
Marius Krauthausen
Michael Emrich
Michael T Heneka
Simon Saxe
P2860
P2888
P304
P356
10.1007/S12035-016-0368-3
P577
2017-01-13T00:00:00Z