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Vaccination Strategies for Alzheimer???s DiseaseFunctional binding interaction identified between the axonal CAM L1 and members of the ERM familyA Case for Microtubule Vulnerability in Amyotrophic Lateral Sclerosis: Altered Dynamics During DiseaseERMs colocalize transiently with L1 during neocortical axon outgrowthBinding partners L1 cell adhesion molecule and the ezrin-radixin-moesin (ERM) proteins are involved in development and the regenerative response to injury of hippocampal and cortical neuronsMicrofluidic culture platform for studying neuronal response to mild to very mild axonal stretch injury.Cyclosporin-A treatment attenuates delayed cytoskeletal alterations and secondary axotomy following mild axonal stretch injury.Stainless steel pinholes for fast fabrication of high-performance microchip electrophoresis devices by CO2 laser ablation.Identifying the primary site of pathogenesis in amyotrophic lateral sclerosis - vulnerability of lower motor neurons to proximal excitotoxicity.Intrinsic regenerative ability of mature CNS neurons.Does beta-amyloid plaque formation cause structural injury to neuronal processes?Mild and repetitive very mild axonal stretch injury triggers cystoskeletal mislocalization and growth cone collapse.Inducing Chronic Excitotoxicity in the Mouse Spinal Cord to Investigate Lower Motor Neuron Degeneration.Neurites containing the neurofilament-triplet proteins are selectively vulnerable to cytoskeletal pathology in Alzheimer's disease and transgenic mouse models.Axonopathy and cytoskeletal disruption in degenerative diseases of the central nervous system.Calretinin and Neuropeptide Y interneurons are differentially altered in the motor cortex of the SOD1G93A mouse model of ALS.Inhibitory dysfunction in amyotrophic lateral sclerosis: future therapeutic opportunities.Mild Traumatic Brain Injury Leads to Decreased Inhibition and a Differential Response of Calretinin Positive Interneurons in the Injured Cortex.Excitotoxicity mediated by non-NMDA receptors causes distal axonopathy in long-term cultured spinal motor neurons.Microfluidic primary culture model of the lower motor neuron-neuromuscular junction circuit.Cortical murine neurons lacking the neurofilament light chain protein have an attenuated response to injury in vitro.The microtubule-stabilizing drug Epothilone D increases axonal sprouting following transection injury in vitro.The degree of astrocyte activation in multiple system atrophy is inversely proportional to the distance to α-synuclein inclusions.Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.Synapse Dysfunction of Layer V Pyramidal Neurons Precedes Neurodegeneration in a Mouse Model of TDP-43 Proteinopathies.Initial calcium release from intracellular stores followed by calcium dysregulation is linked to secondary axotomy following transient axonal stretch injury.Neurofilament triplet proteins are restricted to a subset of neurons in the rat neocortex.Direct determination of the proportion of intra- and extra-cellular neocortical neurofibrillary tangles in Alzheimer's disease.Disruption of the ubiquitin proteasome system following axonal stretch injury accelerates progression to secondary axotomy.Cytoskeletal alterations differentiate presenilin-1 and sporadic Alzheimer's disease.α-Synuclein protects neurons from apoptosis downstream of free-radical production through modulation of the MAPK signalling pathway.Neuroprotective upregulation of endogenous α-synuclein precedes ubiquitination in cultured dopaminergic neurons.Neurochemical diversity of dystrophic neurites in the early and late stages of Alzheimer's disease.Acute reactive and regenerative changes in mature cortical axons following injury.Focal demyelination in Alzheimer's disease and transgenic mouse models.Identification and characterization of a population of motile neurons in long-term cortical culture.Rho kinase activates ezrin-radixin-moesin (ERM) proteins and mediates their function in cortical neuron growth, morphology and motility in vitro.Amyloid β precursor protein regulates neuron survival and maturation in the adult mouse brain.Localization of glutamate receptors in developing cortical neurons in culture and relationship to susceptibility to excitotoxicity.Cytoplasmic cytochrome c immunolabelling in dystrophic neurites in Alzheimer's disease.
P50
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
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հետազոտող
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name
Dickson TC
@ast
Dickson TC
@es
Dickson TC
@nl
Dickson TC
@sl
Tracey C. Dickson
@en
type
label
Dickson TC
@ast
Dickson TC
@es
Dickson TC
@nl
Dickson TC
@sl
Tracey C. Dickson
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Dickson T
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Dickson TC
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prefLabel
Dickson TC
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Dickson TC
@es
Dickson TC
@nl
Dickson TC
@sl
Tracey C. Dickson
@en
P106
P21
P31
P496
0000-0002-9196-1661
P569
2000-01-01T00:00:00Z