Microglia/macrophage polarization dynamics reveal novel mechanism of injury expansion after focal cerebral ischemia. - Wikidata - awgldk - TriplyDB

Microglia/macrophage polarization dynamics reveal novel mechanism of injury expansion after focal cerebral ischemia.

Microglia/macrophage polarization dynamics reveal novel mechanism of injury expansion after focal cerebral ischemia.

http://www.wikidata.org/entity/Q48394793

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The impact of high and low dose ionising radiation on the central nervous system Ischemia, Immunosuppression and Infection--Tackling the Predicaments of Post-Stroke Complications Melatonin and Ischemic Stroke: Mechanistic Roles and Action Neuroinflammation in Parkinson's disease and its potential as therapeutic target The Yin and Yang of innate immunity in stroke Rational modulation of the innate immune system for neuroprotection in ischemic stroke Mechanisms and potential therapeutic applications of microglial activation after brain injury Pathophysiology and neuroprotection of global and focal perinatal brain injury: lessons from animal models Inflammatory responses in hypoxic ischemic encephalopathy Neurorestorative Therapy of Stroke in Type 2 Diabetes Mellitus Rats Treated With Human Umbilical Cord Blood Cells.Intercellular cross-talk in intracerebral hemorrhage Inflammation after Ischemic Stroke: The Role of Leukocytes and Glial Cells Inhibition of prostaglandin E2 receptor EP3 mitigates thrombin-induced brain injury Astrocytes, but not microglia, are activated in oxaliplatin and bortezomib-induced peripheral neuropathy in the rat In vivo molecular markers for pro-inflammatory cytokine M1 stage and resident microglia in trimethyltin-induced hippocampal injury Direct angiotensin AT2 receptor stimulation using a novel AT2 receptor agonist, compound 21, evokes neuroprotection in conscious hypertensive rats FAM19A3, a novel secreted protein, modulates the microglia/macrophage polarization dynamics and ameliorates cerebral ischemia Ethyl pyruvate protects against blood-brain barrier damage and improves long-term neurological outcomes in a rat model of traumatic brain injury Overview of Traumatic Brain Injury: An Immunological Context Pair housing reverses post-stroke depressive behavior in mice.Selective neuronal loss in ischemic stroke and cerebrovascular disease CD200 restrains macrophage attack on oligodendrocyte precursors via toll-like receptor 4 downregulation.Influence of extracellular zinc on M1 microglial activation.Amelioration of amyloid-β-induced deficits by DcR3 in an Alzheimer's disease model.Alterations in the inflammatory cytokines and brain-derived neurotrophic factor contribute to depression-like phenotype after spared nerve injury: improvement by ketamine.Proinflammatory-activated glioma cells induce a switch in microglial polarization and activation status, from a predominant M2b phenotype to a mixture of M1 and M2a/B polarized cells.Recombinant tissue plasminogen activator enhances microglial cell recruitment after stroke in mice Molecular dialogs between the ischemic brain and the peripheral immune system: dualistic roles in injury and repair Old Maids: Aging and Its Impact on Microglia Function.Targeting resolution of neuroinflammation after ischemic stroke with a lipoxin A4 analog: Protective mechanisms and long-term effects on neurological recovery.CD74-immunoreactive activated M1 microglia are shown late in the gerbil hippocampal CA1 region following transient cerebral ischemia.Role of microglia in a mouse model of paediatric traumatic brain injury NOSH-NBP, a Novel Nitric Oxide and Hydrogen Sulfide- Releasing Hybrid, Attenuates Ischemic Stroke-Induced Neuroinflammatory Injury by Modulating Microglia Polarization Influence of Estrogen Modulation on Glia Activation in a Murine Model of Parkinson's Disease Pinocembrin protects hemorrhagic brain primarily by inhibiting toll-like receptor 4 and reducing M1 phenotype microglia.Neuroinflammation and M2 microglia: the good, the bad, and the inflamed.TREM2 protects against cerebral ischemia/reperfusion injury Targeting microglial activation in stroke therapy: pharmacological tools and gender effects.Mechanisms underlying neuro-inflammation and neurodevelopmental toxicity in the mouse neocortex following prenatal exposure to ethanol Therapeutically targeting neuroinflammation and microglia after acute ischemic stroke.

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P2860

Microglia/macrophage polarization dynamics reveal novel mechanism of injury expansion after focal cerebral ischemia.

http://www.wikidata.org/entity/Q48394793

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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name

Microglia/macrophage polarizat ...... after focal cerebral ischemia.

@en

Microglia/macrophage polarizat ...... after focal cerebral ischemia.

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type

label

Microglia/macrophage polarizat ...... after focal cerebral ischemia.

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Microglia/macrophage polarizat ...... after focal cerebral ischemia.

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prefLabel

Microglia/macrophage polarizat ...... after focal cerebral ischemia.

@en

Microglia/macrophage polarizat ...... after focal cerebral ischemia.

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Microglia/macrophage polarizat ...... after focal cerebral ischemia

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Haiying Wang

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Jun Chen

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Peiying Li

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Songela Chen

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Xiaoming Hu

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Yanling Guo

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Yanqin Gao

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3063-3070

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scholarly article

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10.1161/STROKEAHA.112.659656

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2012-08-28T00:00:00Z

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cerebral infarction