Cuprizone induces similar demyelination in male and female C57BL/6 mice and results in disruption of the estrous cycle.
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Dietary vitamin D3 supplements reduce demyelination in the cuprizone modelDefective autoimmune regulator-dependent central tolerance to myelin protein zero is linked to autoimmune peripheral neuropathyGas6 increases myelination by oligodendrocytes and its deficiency delays recovery following cuprizone-induced demyelinationProtective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating diseaseProgesterone and nestorone promote myelin regeneration in chronic demyelinating lesions of corpus callosum and cerebral cortexReduced axonopathy and enhanced remyelination after chronic demyelination in fibroblast growth factor 2 (Fgf2)-null mice: differential detection with diffusion tensor imaging.Demyelination and remyelination in anatomically distinct regions of the corpus callosum following cuprizone intoxicationAn initial top-down proteomic analysis of the standard cuprizone mouse model of multiple sclerosisAttenuation of corpus callosum axon myelination and remyelination in the absence of circulating sex hormonesThyroid hormone alleviates demyelination induced by cuprizone through its role in remyelination during the remission period.Cuprizone demyelination induces a unique inflammatory response in the subventricular zoneGlial response during cuprizone-induced de- and remyelination in the CNS: lessons learned.Estrogens, Neuroinflammation, and Neurodegeneration.Leukemia/lymphoma-related factor (LRF) exhibits stage- and context-dependent transcriptional controls in the oligodendrocyte lineage and modulates remyelination.Clemastine rescues behavioral changes and enhances remyelination in the cuprizone mouse model of demyelination.Activation of the unfolded protein response enhances motor recovery after spinal cord injury.Developmental regulation of group I metabotropic glutamate receptors in the premature brain and their protective role in a rodent model of periventricular leukomalacia.Abbreviated exposure to cuprizone is sufficient to induce demyelination and oligodendrocyte loss.The cuprizone model: regional heterogeneity of pathology.Differential local tissue permissiveness influences the final fate of GPR17-expressing oligodendrocyte precursors in two distinct models of demyelination.Lipopolysaccharide Upregulates the Expression of CINC-3 and LIX in Primary NG2 Cells.
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P2860
Cuprizone induces similar demyelination in male and female C57BL/6 mice and results in disruption of the estrous cycle.
description
2010 nî lūn-bûn
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
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2010年學術文章
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2010年學術文章
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name
Cuprizone induces similar demy ...... sruption of the estrous cycle.
@en
Cuprizone induces similar demy ...... sruption of the estrous cycle.
@nl
type
label
Cuprizone induces similar demy ...... sruption of the estrous cycle.
@en
Cuprizone induces similar demy ...... sruption of the estrous cycle.
@nl
prefLabel
Cuprizone induces similar demy ...... sruption of the estrous cycle.
@en
Cuprizone induces similar demy ...... sruption of the estrous cycle.
@nl
P2093
P2860
P356
P1476
Cuprizone induces similar demy ...... sruption of the estrous cycle.
@en
P2093
Glenn K Matsushima
Jenny P-Y Ting
Lorelei C Taylor
Wendy Gilmore
P2860
P304
P356
10.1002/JNR.22215
P577
2010-02-01T00:00:00Z