Vascular endothelial growth factor is expressed in multiple sclerosis plaques and can induce inflammatory lesions in experimental allergic encephalomyelitis rats.
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Chondroitin sulfate proteoglycans in the nervous system: inhibitors to repairWhite matter hemodynamic abnormalities precede sub-cortical gray matter changes in multiple sclerosisPlasma biomarkers discriminate clinical forms of multiple sclerosisThe expression of VEGF-A is down regulated in peripheral blood mononuclear cells of patients with secondary progressive multiple sclerosisCytokine-mediated blood brain barrier disruption as a conduit for cancer/chemotherapy-associated neurotoxicity and cognitive dysfunction.Gene expression analysis of the microvascular compartment in multiple sclerosis using laser microdissected blood vessels.IFN-β alters neurotrophic factor expression in T cells isolated from multiple sclerosis patients - implication of novel neurotensin/NTSR1 pathway in neuroprotectionCD8 T cell-initiated vascular endothelial growth factor expression promotes central nervous system vascular permeability under neuroinflammatory conditions.Vascular pathology in multiple sclerosis: mind boosting or myth busting?Pathogenic implications of iron accumulation in multiple sclerosis.Angiogenesis in multiple sclerosis and experimental autoimmune encephalomyelitis.Acute demyelinating lesions with restricted diffusion in multiple sclerosis.Sustained expression of vascular endothelial growth factor and angiopoietin-1 improves blood-spinal cord barrier integrity and functional recovery after spinal cord injuryAngiogenesis is present in experimental autoimmune encephalomyelitis and pro-angiogenic factors are increased in multiple sclerosis lesions.Extensive vascular remodeling in the spinal cord of pre-symptomatic experimental autoimmune encephalomyelitis mice; increased vessel expression of fibronectin and the α5β1 integrin.The role of angiogenesis in the pathology of multiple sclerosis.Neuropilin 1 deficiency on CD4+Foxp3+ regulatory T cells impairs mouse melanoma growth.Murine experimental autoimmune encephalomyelitis is diminished by treatment with the angiogenesis inhibitors B20-4.1.1 and angiostatin (K1-3)Soluble VCAM-1 impairs human brain endothelial barrier integrity via integrin α-4-transduced outside-in signalling.Astrocyte-derived VEGF-A drives blood-brain barrier disruption in CNS inflammatory disease.Inhibition of Vascular Endothelial Growth Factor Receptor 2 Exacerbates Loss of Lower Motor Neurons and Axons during Experimental Autoimmune Encephalomyelitis.Mesenchymal stem cells stabilize the blood-brain barrier through regulation of astrocytesHypertonic saline alleviates experimentally induced cerebral oedema through suppression of vascular endothelial growth factor and its receptor VEGFR2 expression in astrocytes.Astrocytic TYMP and VEGFA drive blood-brain barrier opening in inflammatory central nervous system lesionsUpregulation of vascular endothelial growth factor receptor-3 in the spinal cord of Lewis rats with experimental autoimmune encephalomyelitisThe therapeutic potential of neural stem cells.Vascular endothelial growth factor (VEGF) in autoimmune diseases.Endogenous erythropoietin as part of the cytokine network in the pathogenesis of experimental autoimmune encephalomyelitis.VEGF-mediated disruption of endothelial CLN-5 promotes blood-brain barrier breakdown.Hypoxia-ischemia and retinal ganglion cell damage.Enhancing the ability of experimental autoimmune encephalomyelitis to serve as a more rigorous model of multiple sclerosis through refinement of the experimental designAlterations in blood-brain barrier ICAM-1 expression and brain microglial activation after lambda-carrageenan-induced inflammatory pain.VEGF ligands and receptors: implications in neurodevelopment and neurodegeneration.Receptor Tyrosine Kinase and Tyrosine Kinase Inhibitors: New Hope for Success in Multiple Sclerosis TherapyTyrosine Kinase Inhibitor as a new Therapy for Ischemic Stroke and other Neurologic Diseases: is there any Hope for a Better Outcome?Glial regulation of the blood-brain barrier in health and disease.Microglia-blood vessel interactions: a double-edged sword in brain pathologies.VEGF receptor antagonist Cyclo-VEGI reduces inflammatory reactivity and vascular leakiness and is neuroprotective against acute excitotoxic striatal insult.Soluble vascular endothelial growth factor (VEGF) receptor-1 inhibits migration of human monocytic THP-1 cells in response to VEGF.Luteolin inhibits myelin basic protein-induced human mast cell activation and mast cell-dependent stimulation of Jurkat T cells.
P2860
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P2860
Vascular endothelial growth factor is expressed in multiple sclerosis plaques and can induce inflammatory lesions in experimental allergic encephalomyelitis rats.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Vascular endothelial growth fa ...... lergic encephalomyelitis rats.
@en
Vascular endothelial growth fa ...... lergic encephalomyelitis rats.
@nl
type
label
Vascular endothelial growth fa ...... lergic encephalomyelitis rats.
@en
Vascular endothelial growth fa ...... lergic encephalomyelitis rats.
@nl
prefLabel
Vascular endothelial growth fa ...... lergic encephalomyelitis rats.
@en
Vascular endothelial growth fa ...... lergic encephalomyelitis rats.
@nl
P2093
P2860
P356
P1476
Vascular endothelial growth fa ...... lergic encephalomyelitis rats.
@en
P2093
Edward H Oldfield
Marsha J Merrill
Martin A Proescholdt
Nancy Tresser
Steven Jacobson
P2860
P304
P356
10.1093/JNEN/61.10.914
P577
2002-10-01T00:00:00Z