Cholecystokinin-induced c-fos expression in the rat brain stem is influenced by vagal nerve integrity.
about
Upper gastrointestinal dysmotility after spinal cord injury: is diminished vagal sensory processing one culprit?Feeding-dependent activation of enteric cells and sensory neurons by lymphatic fluid: evidence for a neurolymphocrine system.Gene regulation in the magnocellular hypothalamo-neurohypophysial system.Experimental spinal cord injury in rats diminishes vagally-mediated gastric responses to cholecystokinin-8s.The ontogeny of postingestive inhibitory stimuli: examining the role of CCK.Gastrointestinal hormones and the dialogue between gut and brain.Characterization of the feeding inhibition and neural activation produced by dorsomedial hypothalamic cholecystokinin administration.Neurophysiology of hunger and satiety.Functional organization of neuronal and humoral signals regulating feeding behavior.Plasticity in vagal afferent neurones during feeding and fasting: mechanisms and significance.Neural integration of tissue and whole-animal regulation of digestive secretions.Duodenal loading with glucose induces fos expression in rat brain: selective blockade by devazepide.Acid inhibition by intestinal nutrients mediated by CCK-A receptors but not plasma CCK.Neuronal activation and corticotropin-releasing hormone expression in the brain of obese (fa/fa) and lean (fa/?) Zucker rats in response to refeeding.CCK-induced inhibition of presympathetic vasomotor neurons: dependence on subdiaphragmatic vagal afferents and central NMDA receptors in the rat.Hypophagia induced by glucocorticoid deficiency is associated with an increased activation of satiety-related responses.CCK enhances response to gastric distension by acting on capsaicin-insensitive vagal afferents.mu-Opiate receptor agonist loperamide blocks bethanechol-induced gallbladder contraction, despite higher cholecystokinin plasma levels in man.Evidence that cholecystokinin receptors are not involved in the hypothalamic-pituitary-adrenal response to intraperitoneal administration of interleukin-1beta.Gastric distention induces c-fos immunoreactivity in the rat brain stem.Responses to cholecystokinin in the ventromedial nucleus of the rat hypothalamus in vivo.Cytoarchitectonic analysis of Fos-immunoreactivity in brainstem neurones following visceral stimuli in conscious rats.Endogenous cholecystokinin reduces food intake and increases Fos-like immunoreactivity in the dorsal vagal complex but not in the myenteric plexus by CCK1 receptor in the adult rat.Cholecystokinin selectively affects presympathetic vasomotor neurons and sympathetic vasomotor outflow.Meal-related stimuli differentially induce c-Fos activation in the nucleus of the solitary tract.
P2860
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P2860
Cholecystokinin-induced c-fos expression in the rat brain stem is influenced by vagal nerve integrity.
description
1992 nî lūn-bûn
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1992年の論文
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1992年学术文章
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1992年学术文章
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1992年学术文章
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1992年学术文章
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1992年学术文章
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name
Cholecystokinin-induced c-fos ...... nced by vagal nerve integrity.
@en
Cholecystokinin-induced c-fos ...... nced by vagal nerve integrity.
@nl
type
label
Cholecystokinin-induced c-fos ...... nced by vagal nerve integrity.
@en
Cholecystokinin-induced c-fos ...... nced by vagal nerve integrity.
@nl
prefLabel
Cholecystokinin-induced c-fos ...... nced by vagal nerve integrity.
@en
Cholecystokinin-induced c-fos ...... nced by vagal nerve integrity.
@nl
P1476
Cholecystokinin-induced c-fos ...... nced by vagal nerve integrity.
@en
P2093
P304
P356
10.1113/EXPPHYSIOL.1992.SP003579
P577
1992-01-01T00:00:00Z