Pregnancy mitigates cardiac pathology in a mouse model of left ventricular pressure overload.

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Guidelines for Measuring Cardiac Physiology in Mice.Thinking Outside the Box.

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Pregnancy mitigates cardiac pathology in a mouse model of left ventricular pressure overload.

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2016 nî lūn-bûn

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Pregnancy mitigates cardiac pa ...... ventricular pressure overload.

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Pregnancy mitigates cardiac pa ...... ventricular pressure overload.

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Pregnancy mitigates cardiac pa ...... ventricular pressure overload.

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Pregnancy mitigates cardiac pa ...... ventricular pressure overload.

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Pregnancy mitigates cardiac pa ...... ventricular pressure overload.

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Pregnancy mitigates cardiac pa ...... ventricular pressure overload.

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Pregnancy mitigates cardiac pa ...... ventricular pressure overload.

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Bronwen R Herbert

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Dirk J Duncker

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Els Moltzer

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Elza D van Deel

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Heleen van Beusekom

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Hong Xu

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Jolien W Roos-Hesselink

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Mark R Johnson

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Petra Opić

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Suren R Sooranna

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Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia

The AP-1 transcription factor c-Jun prevents stress-imposed maladaptive remodeling of the heart.

The dual-specificity phosphatase MKP-1 limits the cardiac hypertrophic response in vitro and in vivo.

Female sex and estrogen receptor-beta attenuate cardiac remodeling and apoptosis in pressure overload.

Prevention of cardiac hypertrophy by calcineurin inhibition: hope or hype?

Estrogen attenuates chronic volume overload induced structural and functional remodeling in male rat hearts.

An association between gene expression and better survival in female mice following myocardial infarction.

Distinct cardiac transcriptional profiles defining pregnancy and exercise

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scholarly article

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10.1152/AJPHEART.00056.2016

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311

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2016-07-01T00:00:00Z

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27371681

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Pregnancy mitigates cardiac pathology in a mouse model of left ventricular pressure overload.

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Pregnancy mitigates cardiac pathology in a mouse model of left ventricular pressure overload.

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