Inhibition of glycogen synthase kinase enhances isoflurane-induced protection against myocardial infarction during early reperfusion in vivo.

Inhibition of glycogen synthase kinase enhances isoflurane-induced protection against myocardial infarction during early reperfusion in vivo.

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http://www.wikidata.org/entity/Q48706363
Isoflurane protects cardiomyocytes and mitochondria by immediate and cytosol-independent action at reperfusion.Sevoflurane postconditioning reduces myocardial reperfusion injury in rat isolated hearts via activation of PI3K/Akt signaling and modulation of Bcl-2 family proteinsMitochondrial targets for volatile anesthetics against cardiac ischemia-reperfusion injuryCyclosporin variably and inconsistently reduces infarct size in experimental models of reperfused myocardial infarction: a systematic review and meta-analysis.Inhibition of glycogen synthase kinase or the apoptotic protein p53 lowers the threshold of helium cardioprotection in vivo: the role of mitochondrial permeability transitionPharmacologic therapeutics for cardiac reperfusion injury.Does inhibition of glycogen synthase kinase protect in mice?Role of glycogen synthase kinase-3beta in cardioprotection.Volatile anaesthetics and cardioprotection: lessons from animal studies.Cellular signaling pathways and molecular mechanisms involving inhalational anesthetics-induced organoprotection.Wnt/Glycogen Synthase Kinase 3β/β-catenin Signaling Activation Mediated Sevoflurane Preconditioning-induced Cardioprotection.Age-associated differences in activation of Akt/GSK-3beta signaling pathways and inhibition of mitochondrial permeability transition pore opening in the rat heart.Evidence that cardioprotection by postconditioning involves preservation of myocardial opioid content and selective opioid receptor activation.Delayed cardioprotection afforded by the glycogen synthase kinase 3 inhibitor SB-216763 occurs via a KATP- and MPTP-dependent mechanism at reperfusion.The allosteric glycogen synthase kinase-3 inhibitor NP12 limits myocardial remodeling and promotes angiogenesis in an acute myocardial infarction model.Cardioprotection of the aged rat heart by GSK-3beta inhibitor is attenuated: age-related changes in mitochondrial permeability transition pore modulation.Role of MEK, PI3, p38, Tyrosine, and mTOR Kinases in Regulation of Heart Resistance to the Arrhythmogenic Action of Short-Term Ischemia and Reperfusion.
P2860

Inhibition of glycogen synthase kinase enhances isoflurane-induced protection against myocardial infarction during early reperfusion in vivo.

Item
http://www.wikidata.org/entity/Q48706363
description
2006 nî lūn-bûn
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2006年の論文
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2006年学术文章
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2006年学术文章
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name
Inhibition of glycogen synthas ...... ing early reperfusion in vivo.
@en
Inhibition of glycogen synthas ...... ing early reperfusion in vivo.
@nl
type
label
Inhibition of glycogen synthas ...... ing early reperfusion in vivo.
@en
Inhibition of glycogen synthas ...... ing early reperfusion in vivo.
@nl
prefLabel
Inhibition of glycogen synthas ...... ing early reperfusion in vivo.
@en
Inhibition of glycogen synthas ...... ing early reperfusion in vivo.
@nl
P1433
P1476
P2093
P304
P31
P356
P407
P433
P478
P577
P5875
P698
P921
P356
P1433
P1476
Inhibition of glycogen synthas ...... ing early reperfusion in vivo.
@en
P2093
P304
P31
P356
10.1213/01.ANE.0000202379.61338.37
P407
P433
P478
P577
P5875
P698
P921