Evaluation of Akt/mTOR activity in muscle atrophy after rotator cuff tears in a rat model. - Wikidata - awgldk - TriplyDB

Evaluation of Akt/mTOR activity in muscle atrophy after rotator cuff tears in a rat model.

Evaluation of Akt/mTOR activity in muscle atrophy after rotator cuff tears in a rat model.

http://www.wikidata.org/entity/Q48721193

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The role of the peripheral and central nervous systems in rotator cuff disease Upregulation of transforming growth factor-β signaling in a rat model of rotator cuff tears.T lymphocytes are not required for the development of fatty degeneration after rotator cuff tear.Architectural and biochemical adaptations in skeletal muscle and bone following rotator cuff injury in a rat model mTOR regulates fatty infiltration through SREBP-1 and PPARγ after a combined massive rotator cuff tear and suprascapular nerve injury in rats Two Distinct Isoforms of Matrix Metalloproteinase-2 Are Associated with Human Delayed Kidney Graft Function.Muscle atrophy and fatty infiltration after an acute rotator cuff repair in a sheep model TGF-β Small Molecule Inhibitor SB431542 Reduces Rotator Cuff Muscle Fibrosis and Fatty Infiltration By Promoting Fibro/Adipogenic Progenitor Apoptosis.Inhibition of prolyl 4-hydroxylase decreases muscle fibrosis following chronic rotator cuff tear.The role of animal models in tendon research.The Rotator Cuff Organ: Integrating Developmental Biology, Tissue Engineering, and Surgical Considerations to Treat Chronic Massive Rotator Cuff Tears.Animal models for rotator cuff repair.Immunobiological factors aggravating the fatty infiltration on tendons and muscles in rotator cuff lesions.Aging-associated exacerbation in fatty degeneration and infiltration after rotator cuff tear.Differential ubiquitin-proteasome and autophagy signaling following rotator cuff tears and suprascapular nerve injury.Matrix metalloproteinase-2 plays a critical role in overload induced skeletal muscle hypertrophy.Matrix metalloproteinase-2 plays a critical role in overload induced skeletal muscle hypertrophy.Rat rotator cuff muscle responds differently from hindlimb muscle to a combined tendon-nerve injury.Muscle Stem Cell Activation in a Mouse Model of Rotator Cuff Injury.Impaired contractile function of the supraspinatus in the acute period following a rotator cuff tear.mTORC1 Signaling is a Critical Regulator of Postnatal Tendon Development.

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Evaluation of Akt/mTOR activity in muscle atrophy after rotator cuff tears in a rat model.

http://www.wikidata.org/entity/Q48721193

description

2012 nî lūn-bûn

@nan

2012年の論文

@ja

2012年学术文章

@wuu

2012年学术文章

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2012年学术文章

@zh-cn

2012年学术文章

@zh-hans

2012年学术文章

@zh-my

2012年学术文章

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2012年學術文章

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2012年學術文章

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name

Evaluation of Akt/mTOR activit ...... tor cuff tears in a rat model.

@en

Evaluation of Akt/mTOR activit ...... tor cuff tears in a rat model.

@nl

type

label

Evaluation of Akt/mTOR activit ...... tor cuff tears in a rat model.

@en

Evaluation of Akt/mTOR activit ...... tor cuff tears in a rat model.

@nl

prefLabel

Evaluation of Akt/mTOR activit ...... tor cuff tears in a rat model.

@en

Evaluation of Akt/mTOR activit ...... tor cuff tears in a rat model.

@nl

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P1433

Journal of Orthopaedic Research

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Evaluation of Akt/mTOR activity in muscle atrophy after rotator cuff tears in a rat model

@en

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Brian T Feeley

http://www.w3.org/2001/XMLSchema#string

David H Lovett

http://www.w3.org/2001/XMLSchema#string

Dominique Laron

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Hubert T Kim

http://www.w3.org/2001/XMLSchema#string

Sanjum P Samagh

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Sunil K Joshi

http://www.w3.org/2001/XMLSchema#string

Xuhui Liu

http://www.w3.org/2001/XMLSchema#string

Yu-Xuan Dang

http://www.w3.org/2001/XMLSchema#string

P2860

Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo

Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy

Natural history of asymptomatic rotator cuff tears: a longitudinal analysis of asymptomatic tears detected sonographically.

Mechanical stimuli of skeletal muscle: implications on mTOR/p70s6k and protein synthesis.

Expression of atrophy mRNA relates to tendon tear size in supraspinatus muscle.

Suprascapular neuropathy in a shoulder referral practice.

Reversal of suprascapular neuropathy following arthroscopic repair of massive supraspinatus and infraspinatus rotator cuff tears.

Atrophy of S6K1(-/-) skeletal muscle cells reveals distinct mTOR effectors for cell cycle and size control.

Regulation of muscle protein degradation: coordinated control of apoptotic and ubiquitin-proteasome systems by phosphatidylinositol 3 kinase.

The IGF-1/PI3K/Akt pathway prevents expression of muscle atrophy-induced ubiquitin ligases by inhibiting FOXO transcription factors.

P304

1440-1446

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scholarly article

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10.1002/JOR.22096

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9

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30

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2012-02-29T00:00:00Z

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22378614

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