Endothelial deletion of protein tyrosine phosphatase-1B protects against pressure overload-induced heart failure in mice.
about
T helper cells with specificity for an antigen in cardiomyocytes promote pressure overload-induced progression from hypertrophy to heart failure.Protein tyrosine phosphatases in cardiac physiology and pathophysiology.Physiological and pathological gestational cardiac hypertrophy: what can we learn from rodents?Cardiovascular protection by Nox4.The protein tyrosine phosphatase 1B inhibitor MSI-1436 stimulates regeneration of heart and multiple other tissues.Protection against gamma-radiation injury by protein tyrosine phosphatase 1B.
P2860
Endothelial deletion of protein tyrosine phosphatase-1B protects against pressure overload-induced heart failure in mice.
description
2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年學術文章
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2016年學術文章
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name
Endothelial deletion of protei ...... induced heart failure in mice.
@en
Endothelial deletion of protei ...... induced heart failure in mice.
@nl
type
label
Endothelial deletion of protei ...... induced heart failure in mice.
@en
Endothelial deletion of protei ...... induced heart failure in mice.
@nl
prefLabel
Endothelial deletion of protei ...... induced heart failure in mice.
@en
Endothelial deletion of protei ...... induced heart failure in mice.
@nl
P2093
P2860
P356
P1476
Endothelial deletion of protei ...... induced heart failure in mice.
@en
P2093
Astrid Hubert
Katrin Schäfer
Magdalena L Bochenek
Marco R Schroeter
Rajinikanth Gogiraju
Thomas Münzel
P2860
P304
P356
10.1093/CVR/CVW101
P577
2016-05-20T00:00:00Z