Distribution of intraventricularly administered antiamyloid-beta peptide (Abeta) antibody in the mouse brain.

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http://www.wikidata.org/entity/Q48759907

description

2001 nî lūn-bûn

@nan

2001年の論文

@ja

2001年学术文章

@wuu

2001年学术文章

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2001年学术文章

@zh-cn

2001年学术文章

@zh-hans

2001年学术文章

@zh-my

2001年学术文章

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2001年學術文章

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2001年學術文章

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name

Distribution of intraventricul ...... ) antibody in the mouse brain.

@en

Distribution of intraventricularly administered antiamyloid-beta peptide

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type

Item

label

Distribution of intraventricul ...... ) antibody in the mouse brain.

@en

Distribution of intraventricularly administered antiamyloid-beta peptide

@nl

prefLabel

Distribution of intraventricul ...... ) antibody in the mouse brain.

@en

Distribution of intraventricularly administered antiamyloid-beta peptide

@nl

P1476

Distribution of intraventricul ...... ) antibody in the mouse brain.

@en

P2093

Chauhan NB

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Lichtor T

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Siegel GJ

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P2860

Mediated efflux of IgG molecules from brain to blood across the blood-brain barrier.

Monoclonal antibodies inhibit in vitro fibrillar aggregation of the Alzheimer beta-amyloid peptide

Intracerebroventricular injection of anti-Fas activates the hypothalamus-pituitary-adrenal axis and induces peripheral interleukin-6 and serum amyloid A in mice: comparison with other ligands of the tumor necrosis factor/nerve growth factor receptor

Stability, clearance, and disposition of intraventricularly administered oligodeoxynucleotides: implications for therapeutic application within the central nervous system.

Pharmacokinetics and acute toxicology of intraventricular 131 I-monoclonal antibody targeting disialoganglioside in non-human primates.

Peripherally administered antibodies against amyloid beta-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer disease.

Immunohistological study of senile brains by using a monoclonal antibody recognizing beta amyloid precursor protein: significance of granular deposits in relation with senile plaques.

192 immunoglobulin G-saporin produces graded behavioral and biochemical changes accompanying the loss of cholinergic neurons of the basal forebrain and cerebellar Purkinje cells.

192IgG-saporin-induced immunotoxic lesions of cholinergic basal forebrain system differentially affect glutamatergic and GABAergic markers in cortical rat brain regions.

Selective immunolesioning of the basal forebrain cholinergic system disrupts short-term memory in rats.

P304

231-235

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scholarly article

P356

10.1002/JNR.1215

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P433

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P478

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2001-10-01T00:00:00Z

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P698

11592118

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