Short-lived plasma blasts are the main B cell effector subset during the course of multiple sclerosis. - Wikidata - awgldk - TriplyDB

Short-lived plasma blasts are the main B cell effector subset during the course of multiple sclerosis.

Short-lived plasma blasts are the main B cell effector subset during the course of multiple sclerosis.

http://www.wikidata.org/entity/Q48949365

about

Cerebrospinal fluid B cells correlate with early brain inflammation in multiple sclerosis Comprehensive immunophenotyping of cerebrospinal fluid cells in patients with neuroimmunological diseases Borrelia garinii induces CXCL13 production in human monocytes through Toll-like receptor 2 Atacicept: targeting B cells in multiple sclerosis The pathogenesis of lyme neuroborreliosis: from infection to inflammation Neuroinvasion and Inflammation in Viral Central Nervous System Infections B Cells Are Multifunctional Players in Multiple Sclerosis Pathogenesis: Insights from Therapeutic Interventions B Cells in the Multiple Sclerosis Central Nervous System: Trafficking and Contribution to CNS-Compartmentalized Inflammation The Ins and Outs of B Cells in Multiple Sclerosis Diagnostic biomarkers in multiple sclerosis.Screening random peptide libraries with subacute sclerosing panencephalitis brain-derived recombinant antibodies identifies multiple epitopes in the C-terminal region of the measles virus nucleocapsid protein.Prominent plasmacytosis following intravenous immunoglobulin correlates with clinical improvement in Guillain-Barré syndrome.Epstein-Barr virus latent infection and BAFF expression in B cells in the multiple sclerosis brain: implications for viral persistence and intrathecal B-cell activation.Antibodies produced by clonally expanded plasma cells in multiple sclerosis cerebrospinal fluid.Identification of peptide targets in neuromyelitis optica.Intrathecally synthesized IgG in multiple sclerosis cerebrospinal fluid recognizes identical epitopes over time.The antibody genetics of multiple sclerosis: comparing next-generation sequencing to sanger sequencing Systemic inflammation in progressive multiple sclerosis involves follicular T-helper, Th17- and activated B-cells and correlates with progression.Memory B cells from a subset of treatment-naïve relapsing-remitting multiple sclerosis patients elicit CD4(+) T-cell proliferation and IFN-γ production in response to myelin basic protein and myelin oligodendrocyte glycoprotein.Cellular Composition of Cerebrospinal Fluid in HIV-1 Infected and Uninfected Subjects CD19 as a molecular target in CNS autoimmunity.Expansion of CD27high plasmablasts in transverse myelitis patients that utilize VH4 and JH6 genes and undergo extensive somatic hypermutation.The double-edged sword of autoimmunity: lessons from multiple sclerosis.Intrathecal pathogenic anti-aquaporin-4 antibodies in early neuromyelitis optica Uptake and presentation of myelin basic protein by normal human B cells.Update on the autoimmune pathology of multiple sclerosis: B-cells as disease-drivers and therapeutic targets Genetic variants are major determinants of CSF antibody levels in multiple sclerosis.Daclizumab reverses intrathecal immune cell abnormalities in multiple sclerosis Measles virus-specific plasma cells are prominent in subacute sclerosing panencephalitis CSF The Urine Proteome Profile Is Different in Neuromyelitis Optica Compared to Multiple Sclerosis: A Clinical Proteome Study γ-Secretase directly sheds the survival receptor BCMA from plasma cells Immune and Epstein-Barr virus gene expression in cerebrospinal fluid and peripheral blood mononuclear cells from patients with relapsing-remitting multiple sclerosis.Myelin Basic Protein-Induced Production of Tumor Necrosis Factor-α and Interleukin-6, and Presentation of the Immunodominant Peptide MBP85-99 by B Cells from Patients with Relapsing-Remitting Multiple Sclerosis.Epstein-barr virus: environmental trigger of multiple sclerosis?Cerebrospinal fluid B cells from multiple sclerosis patients are subject to normal germinal center selection.Meningeal Infiltration of the Spinal Cord by Non-Classically Activated B Cells is Associated with Chronic Disease Course in a Spontaneous B Cell-Dependent Model of CNS Autoimmune Disease Neurological disorders and therapeutics targeted to surmount the blood-brain barrier The Role of Latently Infected B Cells in CNS Autoimmunity.Dysregulated Epstein-Barr virus infection in the multiple sclerosis brain A Distinct Class of Antibodies May Be an Indicator of Gray Matter Autoimmunity in Early and Established Relapsing Remitting Multiple Sclerosis Patients.

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Short-lived plasma blasts are the main B cell effector subset during the course of multiple sclerosis.

http://www.wikidata.org/entity/Q48949365

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2005 nî lūn-bûn

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Short-lived plasma blasts are ...... course of multiple sclerosis.

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Short-lived plasma blasts are ...... course of multiple sclerosis.

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Short-lived plasma blasts are ...... course of multiple sclerosis.

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Berit Rosche

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Bernhard Hemmer

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Dun Zhou

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Friederike Vogel

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Joachim Sayn

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Norbert Sommer

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Sabine Cepok

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Verena Grummel

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1667-1676

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10.1093/BRAIN/AWH486

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Pt 7

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128

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Hans-Peter Hartung

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2005-03-30T00:00:00Z

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7936043

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15800022

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multiple sclerosis