Melatonin alleviates secondary brain damage and neurobehavioral dysfunction after experimental subarachnoid hemorrhage: possible involvement of TLR4-mediated inflammatory pathway.
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Intracerebral Hemorrhage, Oxidative Stress, and Antioxidant TherapyUtilizing pharmacotherapy and mesenchymal stem cell therapy to reduce inflammation following traumatic brain injuryHigh-Throughput Sequencing and Co-Expression Network Analysis of lncRNAs and mRNAs in Early Brain Injury Following Experimental Subarachnoid HaemorrhageMelatonin-mediated mitophagy protects against early brain injury after subarachnoid hemorrhage through inhibition of NLRP3 inflammasome activation.Melatonin protects against blood-brain barrier damage by inhibiting the TLR4/ NF-κB signaling pathway after LPS treatment in neonatal rats.Roles of Pannexin-1 Channels in Inflammatory Response through the TLRs/NF-Kappa B Signaling Pathway Following Experimental Subarachnoid Hemorrhage in Rats.Orally administered melatonin prevents lipopolysaccharide-induced neural tube defects in mice.HMGB1 in health and disease.Neuroinflammatory responses to traumatic brain injury: etiology, clinical consequences, and therapeutic opportunities.Melatonin attenuates the TLR4-mediated inflammatory response through MyD88- and TRIF-dependent signaling pathways in an in vivo model of ovarian cancerMelatonin attenuates inflammation of acute pulpitis subjected to dental pulp injury.Neuroprotective Mechanisms of Melatonin in Hemorrhagic Stroke.Omega-3 polyunsaturated fatty acid supplementation attenuates microglial-induced inflammation by inhibiting the HMGB1/TLR4/NF-κB pathway following experimental traumatic brain injury.Hydrogen-Rich Saline Attenuated Subarachnoid Hemorrhage-Induced Early Brain Injury in Rats by Suppressing Inflammatory Response: Possible Involvement of NF-κB Pathway and NLRP3 Inflammasome.The NLRP3 Inflammasome: An Important Driver of Neuroinflammation in Hemorrhagic Stroke.Hydrogen Sulfide Ameliorates Early Brain Injury Following Subarachnoid Hemorrhage in Rats.Possible Role of Raf-1 Kinase in the Development of Cerebral Vasospasm and Early Brain Injury After Experimental Subarachnoid Hemorrhage in Rats.DFMG attenuates the activation of macrophages induced by co‑culture with LPC‑injured HUVE‑12 cells via the TLR4/MyD88/NF‑κB signaling pathway.Biochanin A Reduces Inflammatory Injury and Neuronal Apoptosis following Subarachnoid Hemorrhage via Suppression of the TLRs/TIRAP/MyD88/NF-κB Pathway.Neuroinflammation in traumatic brain injury: A chronic response to an acute injuryAntioxidant Melatonin: Potential Functions in Improving Cerebral Autoregulation After Subarachnoid Hemorrhage
P2860
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P2860
Melatonin alleviates secondary brain damage and neurobehavioral dysfunction after experimental subarachnoid hemorrhage: possible involvement of TLR4-mediated inflammatory pathway.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh-hant
name
Melatonin alleviates secondary ...... mediated inflammatory pathway.
@en
Melatonin alleviates secondary ...... mediated inflammatory pathway.
@nl
type
label
Melatonin alleviates secondary ...... mediated inflammatory pathway.
@en
Melatonin alleviates secondary ...... mediated inflammatory pathway.
@nl
prefLabel
Melatonin alleviates secondary ...... mediated inflammatory pathway.
@en
Melatonin alleviates secondary ...... mediated inflammatory pathway.
@nl
P2093
P2860
P356
P1476
Melatonin alleviates secondary ...... mediated inflammatory pathway.
@en
P2093
Chengyuan Ji
Lingyun Wu
Wanchun You
P2860
P304
P356
10.1111/JPI.12087
P577
2013-09-06T00:00:00Z