Effect of prenatal cocaine on dopamine receptor-G protein coupling in mesocortical regions of the rabbit brain.
about
Dopamine receptor activation modulates GABA neuron migration from the basal forebrain to the cerebral cortexThe circadian system is a target and modulator of prenatal cocaine effects.Cocaine exposure modulates dopamine and adenosine signaling in the fetal brain.Specificity of prenatal cocaine exposure effects on cortical interneurons is independent from dopamine D1 receptor co-localization.Characterization of the dopamine receptor system in adult rhesus monkeys exposed to cocaine throughout gestation.Structural, metabolic, and functional brain abnormalities as a result of prenatal exposure to drugs of abuse: evidence from neuroimaging.Sleep deprivation during early-adult development results in long-lasting learning deficits in adult Drosophila.Differential subcellular distribution of rat brain dopamine receptors and subtype-specific redistribution induced by cocaine.Prenatal exposure to drugs: effects on brain development and implications for policy and education.Prenatal cocaine exposure uncouples mGluR1 from Homer1 and Gq Proteins.Executive functioning in preschool-age children prenatally exposed to alcohol, cocaine, and marijuana.Pleiotropic effects of neurotransmission during development: modulators of modularityPrenatal cocaine reduces AMPA receptor synaptic expression through hyperphosphorylation of the synaptic anchoring protein GRIP.Cocaine exposure in utero alters synaptic plasticity in the medial prefrontal cortex of postnatal rats.Drugs, biogenic amine targets and the developing brainCocaine-induced neurodevelopmental deficits and underlying mechanisms.Cocaine exposure decreases GABA neuron migration from the ganglionic eminence to the cerebral cortex in embryonic mice.A transgenic mouse model of neuroepithelial cell specific inducible overexpression of dopamine D1-receptor.Stimulated D(1) dopamine receptors couple to multiple Galpha proteins in different brain regions.An empirical theory of the development of homicide within individuals.Prenatal cocaine/polydrug exposure and infant performance on an executive functioning task.Augmented constitutive CREB expression in the nucleus accumbens and striatum may contribute to the altered behavioral response to cocaine of adult mice exposed to cocaine in utero.Prenatal Cocaine Exposure Alters Signal Transduction in the Brain D1 Dopamine Receptor Systema.Prenatal exposure to cocaine reduces dopaminergic D1-mediated motor function but spares the enhancement of learning by amphetamine in rabbits.Prenatal exposure to cocaine impairs neuronal coding of attention and discriminative learning.Prenatal cocaine exposure: long-term deficits in learning and motor performance.Genetic or pharmacological inactivation of the dopamine D1 receptor differentially alters the expression of regulator of G-protein signalling (Rgs) transcripts.
P2860
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P2860
Effect of prenatal cocaine on dopamine receptor-G protein coupling in mesocortical regions of the rabbit brain.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh-hant
name
Effect of prenatal cocaine on ...... l regions of the rabbit brain.
@en
Effect of prenatal cocaine on ...... l regions of the rabbit brain.
@nl
type
label
Effect of prenatal cocaine on ...... l regions of the rabbit brain.
@en
Effect of prenatal cocaine on ...... l regions of the rabbit brain.
@nl
prefLabel
Effect of prenatal cocaine on ...... l regions of the rabbit brain.
@en
Effect of prenatal cocaine on ...... l regions of the rabbit brain.
@nl
P2093
P1433
P1476
Effect of prenatal cocaine on ...... l regions of the rabbit brain.
@en
P2093
P304
P356
10.1016/S0306-4522(96)83011-9
P407
P577
1996-02-01T00:00:00Z