Systemic levels of IL-23 are strongly associated with disease activity in rheumatoid arthritis but not spondyloarthritis.
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Th17 cells in autoimmune and infectious diseasesImmune-regulatory mechanisms in systemic autoimmune and rheumatic diseasesReview: The interleukin-23/interleukin-17 axis in spondyloarthritis pathogenesis: Th17 and beyondPolymorphic variants of LIGHT (TNF superfamily-14) alter receptor avidity and bioavailability.Tissue specific CD4+ T cell priming determines the requirement for interleukin-23 in experimental arthritisDevelopment of a multi-biomarker disease activity test for rheumatoid arthritisDysregulated serum IL-23 and SIRT1 activity in peripheral blood mononuclear cells of patients with rheumatoid arthritis.Th17 cells expressing KIR3DL2+ and responsive to HLA-B27 homodimers are increased in ankylosing spondylitis.Interleukin 35 Synovial Fluid Levels Are Associated with Disease Activity of Rheumatoid Arthritis.Serum IL-6 and IL-23 Levels and Their Correlation with Angiogenic Cytokines and Disease Activity in Ankylosing Spondylitis, Psoriatic Arthritis, and SAPHO Syndrome.Synovial cytokine expression in psoriatic arthritis and associations with lymphoid neogenesis and clinical featuresAnkylosing spondylitis patients display altered dendritic cell and T cell populations that implicate pathogenic roles for the IL-23 cytokine axis and intestinal inflammationIL-23/Th17 axis is not influenced by TNF-blocking agents in ankylosing spondylitis patientsCCR6 as a possible therapeutic target in psoriasis.Investigating the role of the interleukin-23/-17A axis in rheumatoid arthritisEvidence that autophagy, but not the unfolded protein response, regulates the expression of IL-23 in the gut of patients with ankylosing spondylitis and subclinical gut inflammationProgress in spondylarthritis. Immunopathogenesis of spondyloarthritis: which cells drive disease?Th17 and Th22 cells in psoriatic arthritis and psoriasisDifferences in synovial fluid cytokine levels but not in synovial tissue cell infiltrate between anti-citrullinated peptide/protein antibody-positive and -negative rheumatoid arthritis patients.Synovitis in psoriatic arthritis: immunohistochemistry, comparisons with rheumatoid arthritis, and effects of therapy.Interleukin-23: a key cytokine in inflammatory diseases.Osteoporosis: a paradox in ankylosing spondylitis.Interleukin-23 as a potential therapeutic target for rheumatoid arthritis.Revisiting the old link between infection and autoimmune disease with commensals and T helper 17 cells.Emerging therapies for rheumatoid arthritis.Focus on the potential effects of treatments for spondylarthritides on cardiovascular risk.IL-23 responsive innate-like T cells in spondyloarthritis: the less frequent they are, the more vital they appear.Serum levels of IL-17, IL-4, and INFγ in Serbian patients with early rheumatoid arthritis.The IL-23-IL-17 axis in inflammatory arthritis.Altered immunoregulation in rheumatoid arthritis: the role of regulatory T cells and proinflammatory Th17 cells and therapeutic implications.Lessons to be learned from serum biomarkers in psoriasis and IBD - the potential role in SpA.The Multifunctional Role of the Chemokine System in Arthritogenic Processes.Cytokine profiles in axial spondyloarthritis.Protective role of naturally occurring interleukin-17A-producing γδ T cells in the lung at the early stage of systemic candidiasis in mice.Interleukin-23 in early disease development in rheumatoid arthritis.Synovial effusion and synovial fluid biomarkers in psoriatic arthritis to assess intraarticular tumor necrosis factor-α blockade in the knee joint.Induction of interleukin-23 p19 by serum amyloid A (SAA) in rheumatoid synoviocytes.Senescence marker killer cell lectin-like receptor G1 (KLRG1) contributes to TNF-α production by interaction with its soluble E-cadherin ligand in chronically inflamed joints.IL-23 induces spondyloarthropathy by acting on ROR-γt+ CD3+CD4-CD8- entheseal resident T cells.Synovial fluid and serum levels of IL-17, IL-23, and CCL-20 in rheumatoid arthritis and psoriatic arthritis: a Tunisian cross-sectional study.
P2860
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P2860
Systemic levels of IL-23 are strongly associated with disease activity in rheumatoid arthritis but not spondyloarthritis.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Systemic levels of IL-23 are s ...... tis but not spondyloarthritis.
@en
Systemic levels of IL-23 are s ...... tis but not spondyloarthritis.
@nl
type
label
Systemic levels of IL-23 are s ...... tis but not spondyloarthritis.
@en
Systemic levels of IL-23 are s ...... tis but not spondyloarthritis.
@nl
prefLabel
Systemic levels of IL-23 are s ...... tis but not spondyloarthritis.
@en
Systemic levels of IL-23 are s ...... tis but not spondyloarthritis.
@nl
P2093
P356
P1476
Systemic levels of IL-23 are s ...... tis but not spondyloarthritis.
@en
P2093
Bernard Vandooren
Dirk Elewaut
Elli Kruithof
Herman Mielants
Lode Melis
Martine De Vos
Peggy Jacques
P304
P356
10.1136/ARD.2009.107649
P407
P577
2009-02-05T00:00:00Z