Investigation of the molecular mechanisms underlying postoperative recurrence in prostate cancer by gene expression profiling.

about

Investigation of the molecular mechanisms underlying postoperative recurrence in prostate cancer by gene expression profiling.

Item

http://www.wikidata.org/entity/Q49335670

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Investigation of the molecular ...... by gene expression profiling.

@en

Investigation of the molecular ...... by gene expression profiling.

@nl

type

Item

label

Investigation of the molecular ...... by gene expression profiling.

@en

Investigation of the molecular ...... by gene expression profiling.

@nl

prefLabel

Investigation of the molecular ...... by gene expression profiling.

@en

Investigation of the molecular ...... by gene expression profiling.

@nl

P1476

Investigation of the molecular ...... by gene expression profiling.

@en

P2093

Cheng Yajun

http://www.w3.org/2001/XMLSchema#string

Tang Yuan

http://www.w3.org/2001/XMLSchema#string

Wang Zhong

http://www.w3.org/2001/XMLSchema#string

Xu Bin

http://www.w3.org/2001/XMLSchema#string

P2860

KEGG: kyoto encyclopedia of genes and genomes

The Gene Ontology (GO) project in 2006

Is there a role for IGF-1 in the development of second primary cancers?

MEK5 overexpression is associated with metastatic prostate cancer, and stimulates proliferation, MMP-9 expression and invasion

Human phosphodiesterase 4D7 (PDE4D7) expression is increased in TMPRSS2-ERG-positive primary prostate cancer and independently adds to a reduced risk of post-surgical disease progression

The cAMP phosphodiesterase-4D7 (PDE4D7) is downregulated in androgen-independent prostate cancer cells and mediates proliferation by compartmentalising cAMP at the plasma membrane of VCaP prostate cancer cells

Pathway crosstalk analysis in prostate cancer based on protein-protein network data.

Apigenin attenuates insulin-like growth factor-I signaling in an autochthonous mouse prostate cancer model.

Insulin-like growth factor-1 genotypes and haplotypes influence the survival of prostate cancer patients with bone metastasis at initial diagnosis.

Prediagnostic plasma IGFBP-1, IGF-1 and risk of prostate cancer.

P304

761-768

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.3892/ETM.2017.5510

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-11-14T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29399083

http://www.w3.org/2001/XMLSchema#string

P932

5772610

http://www.w3.org/2001/XMLSchema#string