Distinct Mechanisms of Nuclease-Directed DNA-Structure-Induced Genetic Instability in Cancer Genomes.

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Distinct Mechanisms of Nuclease-Directed DNA-Structure-Induced Genetic Instability in Cancer Genomes.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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Distinct Mechanisms of Nucleas ...... Instability in Cancer Genomes.

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Distinct Mechanisms of Nucleas ...... Instability in Cancer Genomes.

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Distinct Mechanisms of Nucleas ...... Instability in Cancer Genomes.

@en

Distinct Mechanisms of Nucleas ...... Instability in Cancer Genomes.

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Distinct Mechanisms of Nucleas ...... Instability in Cancer Genomes.

@en

Distinct Mechanisms of Nucleas ...... Instability in Cancer Genomes.

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P1476

Distinct Mechanisms of Nucleas ...... Instability in Cancer Genomes.

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P2093

Catherine H Freudenreich

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Changsheng Zhang

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Guliang Wang

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Haihua Zhang

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Imee M Del Mundo

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Jennifer A McKinney

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Junhua Zhao

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Karen M Vasquez

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Pengyu Ren

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Stephen B Boulware

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P2860

XPF with mutations in its conserved nuclease domain is defective in DNA repair but functions in TRF2-mediated telomere shortening

DNA repair by nonhomologous end joining and homologous recombination during cell cycle in human cells

Human XPC-hHR23B interacts with XPA-RPA in the recognition of triplex-directed psoralen DNA interstrand crosslinks.

Advances in mechanisms of genetic instability related to hereditary neurological diseases.

Methods to detect replication-dependent and replication-independent DNA structure-induced genetic instability

Human Flap Endonuclease Structures, DNA Double-Base Flipping, and a Unified Understanding of the FEN1 Superfamily

HADDOCK: a protein-protein docking approach based on biochemical or biophysical information

Phosphorylation of human Fen1 by cyclin-dependent kinase modulates its role in replication fork regulation

Human XPA and RPA DNA repair proteins participate in specific recognition of triplex-induced helical distortions

Repair of damaged DNA by extracts from a xeroderma pigmentosum complementation group A revertant and expression of a protein absent in its parental cell line.

P304

1200-1210

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P31

scholarly article

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10.1016/J.CELREP.2018.01.014

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P433

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P478

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P50

Albino Bacolla

P577

2018-01-01T00:00:00Z

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P698

29386108

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P932

6011834

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