Oxygen Matters: Hypoxia as a Pathogenic Mechanism in Rhinosinusitis.

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Oxygen Matters: Hypoxia as a Pathogenic Mechanism in Rhinosinusitis.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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Oxygen Matters: Hypoxia as a Pathogenic Mechanism in Rhinosinusitis.

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Oxygen Matters: Hypoxia as a Pathogenic Mechanism in Rhinosinusitis.

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Oxygen Matters: Hypoxia as a Pathogenic Mechanism in Rhinosinusitis.

@en

Oxygen Matters: Hypoxia as a Pathogenic Mechanism in Rhinosinusitis.

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Oxygen Matters: Hypoxia as a Pathogenic Mechanism in Rhinosinusitis.

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Oxygen Matters: Hypoxia as a Pathogenic Mechanism in Rhinosinusitis.

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P1476

Oxygen Matters: Hypoxia as a Pathogenic Mechanism in Rhinosinusitis.

@en

P2093

Chang-Hoon Kim

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Hyung-Ju Cho

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P2860

Lung epithelial barrier function and wound healing are decreased by IL-4 and IL-13 and enhanced by IFN-gamma

A nuclear factor induced by hypoxia via de novo protein synthesis binds to the human erythropoietin gene enhancer at a site required for transcriptional activation

Endogenous HMGB1 regulates autophagy

Activation of vascular endothelial growth factor gene transcription by hypoxia-inducible factor 1

High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal

The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

Purification and characterization of hypoxia-inducible factor 1

Depletion of E-cadherin disrupts establishment but not maintenance of cell junctions in Madin-Darby canine kidney epithelial cells.

Role of hypoxia in inflammatory upper airway disease.

Vascular endothelial growth factor produced in nasal glands of perennial allergic rhinitis.

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59-64

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scholarly article

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10.5483/BMBREP.2018.51.2.014

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P433

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P478

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Hyung-Ju Cho

Chang-Hoon Kim

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2018-02-01T00:00:00Z

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P698

29366441

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rhinosinusitis

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5836558

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