Hydrochlorothiazide and acute urinary acidification: The "voltage hypothesis" of ENaC-dependent H+ secretion refuted.

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Hydrochlorothiazide and acute urinary acidification: The "voltage hypothesis" of ENaC-dependent H+ secretion refuted.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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Hydrochlorothiazide and acute ...... ependent H+ secretion refuted.

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Hydrochlorothiazide and acute ...... ependent H+ secretion refuted.

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Hydrochlorothiazide and acute ...... ependent H+ secretion refuted.

@en

Hydrochlorothiazide and acute ...... ependent H+ secretion refuted.

@nl

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Hydrochlorothiazide and acute ...... ependent H+ secretion refuted.

@en

Hydrochlorothiazide and acute ...... ependent H+ secretion refuted.

@nl

P1476

Hydrochlorothiazide and acute ...... dependent H+ secretion refuted

@en

P2093

N Ayasse

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P Berg

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P I A de Bruijn

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P2860

Renal vacuolar H+-ATPase.

Urinary acidification assessed by simultaneous furosemide and fludrocortisone treatment: an alternative to ammonium chloride.

ENaC inhibition stimulates HCl secretion in the mouse cortical collecting duct. II. Bafilomycin-sensitive H+ secretion

Nongenomic stimulation of vacuolar H+-ATPases in intercalated renal tubule cells by aldosterone

Role of proteolysis in the activation of epithelial sodium channels.

Regulation of the epithelial sodium channel by membrane trafficking.

ENaC at the cutting edge: regulation of epithelial sodium channels by proteases.

Surface expression of sodium channels and transporters in rat kidney: effects of dietary sodium.

Physiological role of SLC12 family members in the kidney.

Comparison of bumetanide and hydrochlorothiazide on renal potassium and hydrogen ion excretion.

P304

e13013

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scholarly article

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10.1111/APHA.13013

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223

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Jens Leipziger

M V Sorensen

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2017-12-29T00:00:00Z

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29226589

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